Why MCT Oil Causes Stomach Pain

MCTs burn up and break down fast. They get into our system fast. It’s what makes them so popular. That’s what makes them so unique right now and it’s why they’re so popular with the low-carb craze, simply because they could turn into ketones quick. But this awesome awesome stuff that happens with MCT comes with a little bit of a cost and sometimes that’s cramping, sometimes that’s a little bit of an upset stomach. If you’ve ever consumed MCT oil and you’ve gotten that weird constricting feeling – it almost feels like your intestines are constricting and cramping, then you know exactly what I’m talking about.

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More specifically, you want help with Why MCT Oil Causes Stomach Pain? MCTs burn up and break down fast they get into our system fast. It’s what makes them so popular. What’s what makes them so unique right now. It’s why they’re so popular with the low-carb craze. Simply because they could turn into ketones quick. But this awesome awesome stuff that happens with MCTs comes in a little bit of a cost. Sometimes that’s cramping sometimes that a little bit of an upset stomach. If you’ve ever consumed MCT oil and you’ve gotten that weird constricting feeling it almost feels like your intestines are like constricting and cramping, then you know exactly what I’m talking about. So let’s break down why this happens. But more importantly let’s talk about some solutions that you can use to still get the benefits of MCTs but without the cramping. I’ll give you some ways that you can combine MCTs with other oils and even with some foods that will help change how MCTs treat your body. 

How Fats Are Digested: MCT OIL STOMACH ACHE

In order to understand why MCTs cause a potential problem we have to understand how normal fats are digested. At first because MCTs they’re an anomaly so long chain triglycerides are regular long-chain fats have a pretty complex digestive process. The simple fact of the matter is is that fats are never really digested. They’re moreso just emulsified because if you were to mix up some oil and water it’s never gonna really break down. You can whip it up and get it into really small bubbles but you’re not gonna ever really break it down. Same thing kind of happens in your body until it’s at the cellular level. So what happens is you consume a regular fat, it goes in your stomach and then it drops into your small intestine. When it drops in your small intestine, it triggers the release of a specific thing called cholecystokinin. What this cholecystokinin does is it triggers a cascade of events that break down the fats. So the cck first of all makes us feel satiated so we love it right. It makes us feel full but cck also triggers pancreatic lipase, so it triggers fat breaking down enzyme so enzymes that come from the pancreas that help break down fats. Okay but it also triggers the gallbladder to release bile which breaks down fats. So what happens is the fats start to break down they come in as triglycerides they break down into free fatty acids and then what happens is the bile salts come in and they break down those free fatty acids into something known as micelles. So it breaks them down into these really teeny teeny pieces and they aggregate into micelles. The micelles then enter the cells that are in our small intestine and inside the cell of our small intestine they reformed back to a triglyceride and then get dumped into our lymphatic system where they turn into clio microns and then they finally get broken down again and again and eventually they end up giving us that as a fuel source. Right so eventually they’re gonna get into the mitochondria. The point is there’s a lot going on. It’s not like it’s simply eating it and it gets absorbed. It goes through a multi-phase emulsification system. 


Okay so now we know that complex model now let’s take a look at what happens with MCTs and tell me if you recognize any difference here. I consume MCTs goes into my stomach then it drops into my small intestine. No cholecystokinin is released. No bile is released. It goes in it doesn’t even enter as a micelle. It doesn’t even really enter the entero site. It goes through passive diffusion and enters my body like it enters my bloodstream and then once it enters the bloodstream MCTs are so fast that they go right into the mitochondria like normally our cells require carnitine to let fat in that’s why l carnitine is such a popular fat burning supplement. Because it’s supposed to allow fat to get into the cell better. MCTs don’t even require l carnitine they like have this vip backstage pass to everything in our body where they just go to the front of the line. Think of it like a disneyland fast pass. They just go to the front always. Why they get preferential treatment I have zero idea. The fact is because they’re shorter chains of fat they just have to skip that process. They can just go right in. So this of course comes with a cost. They digest fast so we’re not getting the actual things that help slow them down. They’re just going through passive diffusion and absorb right away. Now the american journal of clinical nutrition published a study that proved this. So what they did is they took a look at six test subjects and they had them consume either sixty millimoles of MCTs or sixty millimoles of long chain triglycerides regular fats and they wanted to measure to see if there was any cck response. Well what they found is those that consumed the long-chain fatty acids that ended up consuming the lc ts. Well they had a reduction in gallbladder size indicating that the gallbladder was releasing bile. But they also had a decent increase in c ck. Okay so they saw a correlation there then with the MCT group. No change in gallbladder. So the gallbladder didn’t release any bile but then. No change in c ck. It didn’t even trigger c ck response. So the downside is that means that you’re not gonna have any satiety. But the other downside is it’s gonna absorb so fast it’s going to give you a cramp and that’s exactly what’s happening. 


So on one hand it’s cool because it’s absorbing fast, on the other hand its cramping you so how do we fix this? Okay the simplest thing is MCT’s straight up are not the greatest. Like there’s no real solid benefit to just taking MCTs without other fats along with it so you might as well consume coconut oil. I mean MCT taste great and they’re easy and they’re quick energy. But coconut oil contains a good amount of MCT’s but they also contain long-chain fats and lauric acid so that means you’re still getting the benefit of the MCTs. The MCTs aren’t attached to the other fat so if I take a spoonful of coconut oil and I eat it, the MCTs are going to be separate from the rest of the fats. The MCTs are still going to absorb super fast I’m still going to get the benefit but at least the long-chain fats from the coconut oil are gonna at least trigger the cholecystokinin. So I’m satiated and they’re also going to trigger the pancreatic lipase and they’re also going to trigger the bile reaction. So we get a win win. We get the MCTs to get absorbed but then we also get the lauric acid and the coconut oil longer chain fats that trigger the other hormonal responses and enzymatic responses. Problem solved win-win we still get the quick energy. 


The other thing that you can do is reduce your fodmap foods. Okay so fodmap stands for fermentable oligosaccharides disaccharides monosaccharides and polyols. So basically what they are there are certain vegetables and some fruits that end up breaking down in a unique way where they basically make these specific sugars that cause bloating. Right well it causes the similar cramping because of passive diffusion with fodmap foods that would occur with MCTs. So if you reduce your fodmap foods your fodmap overall levels you can improve this. But the other thing that I was gonna say is that if you add MCTs into your diet put them over low fodmap foods. So like drizzle them on a little bit of zucchini. That’s one of my favorite things to do. Take some zucchini drizzle some MCT oil on it, put some spices on it, you get the benefit of the MCTs but the overall fiber and the low fodmap nature of the zucchini and stuff like that is gonna make it so you don’t get the cramps as much. So you can google low fodmap foods and get a whole list if you wanted to do that. 


So lastly one of the things you can do is eat more in the way of fermented veggies and fermented foods in general. All this was going to do is gonna help stimulate some more digestion. So these fermented veggies by themselves are gonna trigger the release of different enzymes in and of themselves. Plus you’re adding these enzymes into your diet so at least helps the process and could stop some of the quick breakdown when it comes down to those cramps that you get from the MCT oil. So I know this wasn’t the end-all be-all honestly MCT oil is always going to be MCT oil and the more that we consume it in powder form and stuff like that, the faster we’re absorbing it and the faster these problems are gonna hit us. The thing is it’s not harmful it just makes us a little nauseous and a little crampy. But in time it goes away and you still get the benefit.   This Post Was All About Why MCT Oil Causes Stomach Pain. Why MCT Oil Causes Stomach Pain

Here’s The Video Description From YouTube

Why MCT Oil Causes Stomach Pain: The entry of triglycerides as LCTs from the stomach into the duodenum stimulates the enteric secretion of the hormone cholecystokinin (CCK) and pancreatic enzymes from the pancreas CCK promotes further release of bile from the gallbladder to help emulsify the triglycerides into smaller fat droplets to maximize its digestion The chylomicrons are released via exocytosis, enters and travels through the lymphatic system and eventually, drains into the subclavian vein to reach the bloodstream In the intracellular space, long-chain fatty acids bind to carnitine for transport into the mitochondria for subsequent β-oxidation Note: In carnitine deficiency states that contribute to severe protein malnutrition (e.g., chronic malabsorption, small bowel obstruction, starvation), these long-chain fatty acids cannot be efficiently utilized and instead lead to accumulation of unoxidized fatty acids and impairment of ureogenesis, ketogenesis, and gluconeogenesis. By contrast, MCT digestion is rapid and simple – MCTs do not stimulate CCK secretion MCT absorption occurs via passive diffusion along the gastrointestinal tract into the portal system bound to albumin No further modification of the MCT molecules is required – moreover, MCTs are not dependent on the carnitine acyltransferase system for transport into the mitochondria for β-oxidation This provides the ability for more rapid metabolism of MCTs and improved utilization even in states of protein deficiency Study – The American Journal of Clinical Nutrition The present study was undertaken to compare the effects of equimolar amounts of long-chain triglycerides (LCT) and medium-chain triglycerides (MCT) on plasma cholecystokinin (CCK) concentrations and gallbladder contraction in man On separate mornings and in random order six healthy volunteers ingested either 60 mmol LCT or 60 mmol MCT Plasma CCK concentrations were measured by a sensitive and specific radioimmunoassay and gallbladder contraction by ultrasonography Ingestion of LCT induced significant increases in plasma CCK and decreases in gallbladder volume On the other hand, no significant changes in plasma CCK and gallbladder volume were found after MCT Ingestion of MCT was followed by abdominal cramps and diarrhea, while LCT were without side effects Concluded that, in contrast to LCT, MCT do not induce CCK release and gallbladder contraction Cholecystokinin is the principle stimulus for delivery of pancreatic enzymes and bile into the small intestine Dosage A tablespoon (15 mL) of MCT oil contains 14 grams of fat and 115 calories A maximum daily dose of 50-100 grams has been suggested for improved gastrointestinal tolerance; this is equivalent to 4-7 tablespoons (60-100 mL) per day (56-98 grams of fat and 460-805 calories) The daily dose of MCTs should be increased as tolerated to the maximum daily dose, while equally dividing the dose across all meals Solutions Coconut Oil over MCT Oil Coconut oil is comprised of some LCTs, which stimulate the enteric secretion of the CCK and subsequent pancreatic enzymes from the pancreas As such, CCK promotes further release of bile from the gallbladder to help emulsify the triglycerides into smaller fat droplets to improve digestion Watch FODMAP Intake FODMAP is an acronym that stands for Fermentable Oligosaccharides, Disaccharides, Monosaccharides And Polyols Your body is unable to completely digest these sugar molecules, they travel through your GI tract and reach your colon undigested, where the bacteria that live in your colon begin to ferment them – the fermentation can produce gas and bloating Swapping high FODMAP foods for low FODMAP foods (i.e. spinach, zucchini, etc.) and have reduce GI side effects *Not a direct way to combat digestive issues from MCT oil, but reducing intake can help lessen GI distress and won’t exacerbate the situation* Cooking vegetables helps break down the fibers they contain, which makes them easier on the digestive system because they’re already partly broken down. Resources 1) Effect of equimolar amounts of long-chain triglycerides and medium-chain triglycerides on plasma cholecystokinin and gallbladder contraction. (1984, March 1). Retrieved from 2) Halmos EP , et al. (n.d.). A diet low in FODMAPs reduces symptoms of irritable bowel syndrome. – PubMed – NCBI. Retrieved from 3) 4) Cholecystokinin. (n.d.). Retrieved from l

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