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Reduce Hip and THIGH Fat – Simple Strategies
Researchers used to think overweight people had stronger skeletons and were protected against bone loss as they age
The latest research, however, suggests that fat accumulation is actually associated with weaker bone-mineral density
Actually an Inverse Correlation
A meta analysis published in The Journal of Endocrinology & Metabolism identified 44 studies that had examined the correlation between LM, FM, and BMD between 1989 and 2013
These studies involved 20,226 men and women (4966 men and 15,260 women) aged between 18 and 92 years
It was found that higher FM was correlated with weaker bone mineral density than with LM
Belly Fat & Osteoporosis
A study was presented at the Radiological Society of North America (RSNA) evaluated 35 obese men with a mean age of 34 and a mean body mass index (BMI) of 36.5
The men underwent CT of the abdomen and thigh to assess fat and muscle mass, as well as very high resolution CT of the forearm and a technique called finite element analysis (FEA), in order to assess bone strength and predict fracture risk.
Fat & Bone Marrow
Another study, published in Radiology, found that excess liver and muscle fat (in addition to belly fat) are also detrimental to bone health
This study looked at fat inside bone marrow, the spongy tissue inside the bones of the body that produces stem cells
They focused on bone marrow fat because that is where our stem cells can develop into osteoblasts – the cells responsible for bone formation – or fat cells
Higher levels of bone marrow fat put people at increased risk of fracture
Bone marrow fat makes bones weak – if you have a spine that’s filled with fat, it’s not going to be as strong
Cytokines & Osteoporosis
The mechanism behind this is believed to be due to the inflammatory cytokines that visceral fat releases
Triglycerides, the type of fat found in the blood, also have a positive correlation with bone marrow fat, possibly because they stimulate osteoclasts, a type of cell that breaks up bone tissue
cell-signaling molecules called cytokines are known to promote the conversion of stem cells into fat.
The process of bone remodeling involves complex interactions between the osteoclast, the primary bone-resorbing cells, and other cells in its microenvironment
In contrast, cytokines such as interleukin-4, gamma-interferon, and transforming factor-beta inhibit both osteoclast formation and osteoclast activity
Fat Distribution & Safety
There was a long held the belief that the fat that accumulates around the thighs and butt isn’t as dangerous as the fat that makes up muffin tops and love handles, since the latter contains metabolically active cells that promote insulin resistance and raise diabetes and heart disease risk
High chemerin levels, for example, correlated with high blood pressure, elevated levels of C-reactive protein (a sign of inflammation) and triglycerides, insulin resistance, and low levels of HDL cholesterol.
Low omentin-1 levels correlated with high levels of triglycerides and blood glucose levels and low levels of HDL cholesterol
Simply, they found that fat stored in the buttock area – also known as gluteal adipose tissue – secretes abnormal levels of chemerin and omentin-1, proteins that can lead to inflammation and a prediabetic condition known as insulin resistance
All calories are created equal (carbs make you fat/fat makes you fat)
Researchers investigated liver fat accumulation and body composition during overfeeding saturated fatty acids (SFAs) or polyunsaturated fatty acids (PUFAs)
39 normal-weight individuals were overfed muffins high in SFAs (palm oil) or PUFAs (sunflower oil) for 7 weeks
Liver fat, visceral adipose tissue (VAT), abdominal subcutaneous adipose tissue (SAT), total adipose tissue, pancreatic fat, and lean tissue were assessed
Both groups gained similar weight – SFAs, however, markedly increased liver fat compared with PUFAs and caused a twofold larger increase in VAT than PUFAs
Conversely, PUFAs caused a nearly threefold larger increase in lean tissue than SFAs
Increase in liver fat directly correlated with changes in plasma SFAs and inversely with PUFAs
Concluded that overeating SFAs promotes hepatic and visceral fat storage more so than PUFAs
Carb vs Fat Storage
Point here is that neither carbs nor fat will “make you store fat,” but either one in excess CAN be stored as fat – but fat gets stored more easily