How to Lower Cholesterol: LDL vs. HDL- Thomas DeLauer

How to Lower Cholesterol: LDL vs. HDL- Thomas DeLauer

How to Lower Cholesterol: LDL vs. HDL- Thomas DeLauer

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LDL cholesterol is not bad now I’m prepared to get totally flamed and totally destroyed on this video and that’s absolutely fine but that’s why I’m going to start this video off with an awesome reference an awesome study from the British Medical Journal that is literally brand spanking new and what this study from the British Medical Journal looked at was over 68,000 participants from over nineteen studies what they were trying to identify was if there truly was a correlation between LDL cholesterol and cardiovascular related debt well what they found was pretty insane eighty percent of the people that died did not have a correlation with LDL cholesterol at all in fact was quite the opposite what they found is that those that had lower levels of LDL cholesterol and lower levels of the LDL cholesterol the bad cholesterol had a higher instances of death related to cardiovascular issues that’s Point Blank right there cut and dry so let’s get right in to the science and how this works I’m going to break this video down as simply as I possibly can because I feel that everyone deserves the right to know how cholesterol really works in the body and it’s really not that bad we just have to understand what’s happening so it starts with what is cholesterol cholesterol is a fatty substance that really helps us build almost everything in our body helps us build nerves helps us build cells it helps us create hormones it’s absolutely critical it’s so critical that the body has to create it itself in some cases now something the body has to create itself can’t really be all that bad but we have to break down a little bit more of what a good cholesterol and what a bad cholesterol really is well let me answer that for you there’s no such thing there really is not a good or a bad cholesterol there neither because LDL which is known as the bad cholesterol is really a lipoprotein and HDL which is known as the good cholesterol is also a lipoprotein not a cholesterol and what a lipoprotein is is really just a carrier of cholesterol see think of it like a private chauffeur for cholesterol cholesterol is not water-soluble so what that means is it cannot just flow on through the bloodstream willy-nilly going to where it needs to go it needs specific chauffeurs and specific drivers so we need those lipoproteins to carry cholesterol so LDL HDL I dlv LDL and even some other ones like Clio microns are technically the vehicles that drive the cholesterol not the cholesterol itself think of them like specific kind of ubers buses trains different transportation vehicles for different needs that’s really how the process starts so let’s break down what happens from the time you consume a lipid all the way down to what LDL and what VLDL and what HDL are truly doing in your body when you eat lipids your body starts to digest them okay and it goes through the entero sites which is basically the cell in your intestinal tract that allows the absorption from there it piles on to what’s known as a Clio micron plyo micron is one of those lipoproteins I just talked about and it delivers it to specific places well the main job of the Clio micron is to deliver something known as ta G but you may commonly know it as a triglyceride something that your doctor is telling you to reduce to and quite honestly that’s probably the one thing that they’re correct about triglycerides are seemingly very very bad so the clio micron delivers a little of the triglycerides very very energy rich form of fuel triglyceride jumps off and is used for fuel when need be any excess triglycerides ultimately get converted into fatty acids that either get used for energy or stored as adipose tissue so triglycerides are sort of a precursor to building up that belly fat that we don’t really want okay then we have the LDL which is produced by the liver and also produced by clio microns vldls carries some of the cholesterol but not as much of the clio microns do right out the gate the same general premise delivering the fresh new cholesterol to where it needs to go I’ll come back to LDL in just a second because I’m going to jump over to HDL for a second but the first thing I really want you to know about this is LDLs deliver new cholesterol and HDLs deliver recycled cholesterol here’s how it works so the liver produces HDL the good cholesterol and what this good cholesterol does is it leaves the liver totally empty think of it like an empty bus just the shell of a bus with no people on it it drives to the cell and the cell says hey I have excess cholesterol that I either didn’t use or I have already used and it’s ready to be recycled it hops on the HDL and the HDL drives back to the liver and it ends up doing its job it ends up converting it back into a useful form of cholesterol or ends up excreting it by combining with bile so that’s where HDL comes in as the good cholesterol right seems like a hero it goes through it picks up all the spare cholesterol and it delivers them to where they need to go and you’re never adding new cholesterol you’re just recycling more of a good thing right so then LDLs are the ones that actually carry the cholesterol to the cell to be utilized here’s where it all gets really tricky and here’s where I’m going to try to summarize it as simply as possible LDLs deliver the cholesterol to the cell the new cholesterol to the cell the cell has a receptor specifically for LDLs so remember LDLs aren’t bad so much so that even our cells have receptors for it the LDL hits the receptor and through something known as receptor cell mediated endocytosis it receives the cholesterol and transfers it into the cell then the receptor cell drops up the cholesterol and comes back to the surface of the cell and therefore is readily available again from new cholesterol to be received I’m going to make this abundantly clear it is the sole responsibility of the LDL receptor to regulate the levels of LDL cholesterol in the blood it’s up to the cell to dictate how much is absorbed and how much stays in the blood so here’s what ends up happening and this is where the medical community has made their hypothesis their overall literally known as the cholesterol hypothesis we have receptor cell mediated endocytosis actions happening in the blood vessels – so when we have excess LDL floating through the bloodstream it’s going to bind to those receptor cells in our bloodstream creating sort of an LDL blockage but that’s not the problem what ends up happening is this is somewhat abnormal so our body’s trigger an immune response they send in macrophages they send the immune system to attack that what does our immune system always do when we bump our knee by our elbow inflammation causes swelling well guess what this swelling is happening surrounding the LDL build-up that happened in the artery that causes the inflammation which causes atherosclerosis and there are so many doctors that have tried speaking from the mountaintops to talk about this that truly atherosclerosis is real – inflammation not necessarily cholesterol we have to link back to the fact that it’s took triglycerides that lead to the fat accumulation not the cholesterol and ends up being the LDL communication in the immune system that Lisa atherosclerosis now honestly I’m not a doctor I can’t tell you to go on lipitor I can’t tell you to go on blood pressure medication I can’t tell you to even really eat a low cholesterol or a high cholesterol diet but I can be a purveyor of good things and a purveyor of good information and really sift through the studies that really prove that LDL has such a bad rap it’s really not that bad after all so at the end of the day remember LDL delivers new cholesterol new cholesterol equals new life new hormones new cells new nerves HDL is good – it recycles more of a good thing but they’re both good don’t demonize one or the other they’re both an integral part of our life and they’ve been here through evolution they’ve been here through so many different things for decades for centuries for hundreds of thousands of years and it’s how our bodies work it’s up to us to live a stress-free lifestyle and an anti-inflammatory lifestyle so that we don’t trigger the inflammation from an already existing positive thing in our bodies as always make sure you keep it locked in on my channel and if you have any questions or you just want to flay me and get totally upset with me for doing this video have that comment away I will see you in the next video

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How to Lower Cholesterol: LDL vs. HDL- Thomas DeLauer

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How to Lower Cholesterol: LDL vs. HDL- Thomas DeLauer…
Good vs. Bad Cholesterol:
LDL cholesterol is not bad. HDL is not cholesterol and neither is LDL. “High” and “low” refer to the relative proportion of protein to fat in the bundle. HDL stands for High Density Lipoprotein. LDL stands for Low Density Lipoprotein. (There are three other lipoproteins; chylomicrons, VLDL and IDL) Cholesterol and fat are not water-soluble so they need to be carried around the body in something to do their vital work. The carriers of such substances are called lipoproteins. (4)
Lipoproteins:
Lipoproteins travel round the bloodstream and act as transporters. So LDL and HDL are carriers of cholesterol, as well as triglycerides and phospholipids and proteins. LDL is the carrier of fresh cholesterol and HDL is the carrier of recycled cholesterol.
VLDL, LDL, and HDL:
The liver is central to the regulation of cholesterol levels in the body. Not only does it synthesize cholesterol for export to other cells, but it also removes cholesterol from the body by converting it to bile salts and putting it into the bile where it can be eliminated in the feces. Chylomicrons and VLDL deliver TAG to cells in the body. Two types of lipoproteins are triglyceride-rich: the chylomicrons and VLDL. Chylomicrons are synthesized by enterocytes from lipids absorbed in the small intestine. VLDL is synthesized in the liver. The function of these lipoproteins is to deliver energy-rich triacylglycerol (TAG) to cells in the body (pink pathway). TAG is stripped from chylomicrons and VLDL through the action of lipoprotein lipase, an enzyme that is found on the surface of endothelial cells. HDL is involved in reverse cholesterol transport. Excess cholesterol is eliminated from the body via the liver, which secretes cholesterol in bile or converts it to bile salts. The liver removes LDL and other lipoproteins from the circulation by receptor-mediated endocytosis. It travels in the circulation where it gathers cholesterol to form mature HDL, which then returns the cholesterol to the liver via various pathways. LDL delivers cholesterol to cells in the body. As VLDL particles are stripped of triacylglycerol, they become more dense. These particles are remodeled at the liver and transformed into LDL. The function of LDL is to deliver cholesterol to cells, where it is used in membranes, or for the synthesis of steroid hormones. Cells take up cholesterol by receptor-mediated endocytosis. LDL binds to a specific LDL receptor and is internalized in an endocytic vesicle. Receptors are recycled to the cell surface, while hydrolysis in an endolysosome releases cholesterol for use in the cell. LDL is what we call a carrier protein, and one of its important jobs is to carry a fundamentally important chemical to every cell in the body. This chemical is a critical component of cell membranes, serves as a brain antioxidant, and is the raw material from which your body manufactures vitamin D, cortisol, estrogen, progesterone, and testosterone.
Study – The British Medical Journal (BMJ):
The research team — comprised of experts from seven different countries — evaluated data collected from 19 studies on a total of 68,094 older adults. The team was seeking to determine if LDL cholesterol is associated with death in the older adults. According to the cholesterol hypothesis, it should directly relate. According to the BMJ study, it doesn’t. Researchers say almost 80 percent of the participants in the studies who had high LDL cholesterol did not die because of their cholesterol level. On the other hand, researchers discovered people with low levels of LDL cholesterol, or LDL-C, had the highest rates of death related to cardiovascular disease, the leading cause of death for both men and women in the United States.

References:
1) Cholesterol, lipoproteins and the liver. (n.d.). Retrieved from

2) Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review. – PubMed – NCBI. (n.d.). Retrieved from

3) Plasma lipids and cerebral small vessel disease. – PubMed – NCBI. (n.d.). Retrieved from

4) We have got cholesterol completely wrong – Zoë Harcombe. (n.d.). Retrieved from

5) Why Cholesterol is Not Bad | Paleo Leap. (n.d.). Retrieved from /

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