Do you have the Cholesterol that kills?

Do you have the Cholesterol that kills?

Check out the video on Do you have the Cholesterol that kills?.
Hello everybody it's Dr baz we are here Tuesday night helping you reverse those medical problems with healthy keto living I love seeing where you guys are at so please tell me in the chat where you're from and of course my uh live sound check it's nice to know you can hear and see me I am uh going to pick up where we left off last week with a case.

Study because we're going to talk about the cholesterol topic that I haven't avoided I just know that it's a topic that deserves time and um well frankly most of the people on YouTube don't take time it's this really short quick video we're going to talk about where this we're going to unpack whether or not the cholesterol that you.

Have in your body is it the kind that causes the heart attack or not and the statins deserve this bet of a wrap let's talk about the truth there I do want to revisit uh last week's um last week's um a case that I'm going to give a brief intro on this slide and then we're gonna.

Do some of our other Traditions here as I go to this one here so last week we had this awesome case we had lots of you tuned in for this and I want you to hear a few things that I did not share last week as you look um I'm going to show you uh this one this panel was done October of 22 and this one is November.

Or maybe it was November's day anyway it's 30 days apart no it's right before he did keto and right in this past week where his total cholesterol went from 215 before he did keto to 3 30 after he did keto he had triglycerides of only 45 he started keto and now his triglycerides are up to 128. his good cholesterol started out at 93.

And now it's 66 again wrong direction if you're looking at tradition your his vldl was a puny little nine good number now it's up to 23 and has bad cholesterol was 113 and now is 241 241. there's several other labs in there but the point of showing you Joel's numbers from last week is to give you a hint of.

One thing he uh that I tried to come across last week sharing this with you I'm not sure how good of a job I landed on it but I'm going to drive that point home today and that is Joel is a typical person on the keto diet finding lots of people talking about it and he dove in so fast so quick that his body has not adjusted and the.

One thing the one thing he could have done would have been to instead of clicking around YouTube for hours and hours which he did he tried to educate himself really quickly but there was a smarter way to do this and that is with the online course that I offer and in the next week you get it for free if you do this.

Let me go to uh this so on here yes on my website if you go to store there also should be a pop-up uh it's not going to happen on mine because I'm already here but there's this thing called a Christmas bundle and the Christmas bundle is in honor of people just like Joel who really want to do keto but Joel kind of belongs in my.

Clinic he has had some medical problems that uh he's done his best he's kind of biohacked his way through it and then he went so quick into the keto diet that he he did like he's almost as good of an example to teach from as the person that I that I wrote that book about uh called David who really did everything backwards in this uh bundle not only do.

You get the course for that's 500 bucks you get it for free when you order this bundle um again the whole package is worth about 815 dollars for those of you that have wanted the keto combo these bags are sold out except for in the bundle and uh this is also designed mind for you to gift it now I want you to be a little careful.

That if you gift the keto diet to somebody that you're sending the right message the right message of oh I know you're interested and you're struggling here's the cliff notes uh uh Evelyn says please please move on stop selling Evelyn this is exactly why I do this is the.

Number of people that write into this Channel with stories like Joel's with stories with cholesterol that does this and they wonder why why does this happen Evelyn it's because they skipped the the learning they skipped the education that really does come with the keto diet and that is a powerful mistake that would make me not recommend it if Joel was.

Going to pick the path that he's been on the last month which is the fastest loss of 30 pounds he's lost in a month uh in a way that really did shift his um his chemistry to the potential to be better but long-term he he's in trouble uh he needs to course correct and thank goodness he is here in Tampa he's a snow Burger that's uh here in Florida and.

He's attending our support group that is free because Evelyn there's a lot of things I do for free and there's a few things that I've worked on to help support this channel one of them is tonight you'll notice that I don't have the commercials on I turned them off to say no I really want to not annoy my viewers I really want to connect with.

You and I start by showing you that I don't uh talk about the keto diet without doing it you're not on the keto diet unless you check your numbers and the numbers that I mean aren't just waistline and cholesterol like we are going to talk about tonight but they are your blood ketones and blood glucose numbers looking at the metrics inside.

How people uh how insulin is speaking to you uh to your body how well how sensitive or insensitive your body is in other words how resistant it is has to do with what could I have predicted that Joel was going to have this problem the answer is yes I totally could have um so I have a glucose of 65 and ketones are counting down here.

Um almost there yeah 1.2 so about uh 40 minutes ago my glucose was 59 and my ketones are one point uh they're somewhere in the one I can't remember what they were I am totally going to drink some bubble water with pucker up in it tonight because I have an awesome review that somebody.

Wrote me that I will be reading here in a bit and when I read your review you get a free gift from the DR Boss channel that the ones from last week I just sent out probably um I just sent out uh actually I think I went out today so I'm going to put all.

Of this in here and um the bubbles in here and that's one of my favorite ways to drink this again all pucker up is is liquid ketones it is very sour my husband I have this ongoing bet that uh the the reviews for taste are going.

To be not something I should show off but I I am showing off uh let me go to this over here on my channel under the Dr BOS pucker up I have a great review um by I think it was Teresa's I was supposed to read let me double check on that yes Teresa's stockroom so on December.

9th she writes in a really great review and she said I received my pucker up can you see it I can make it a little bigger I received my pucker up yesterday and could not wait to try it I have to tell you it tastes fabulous uh I'm only snickering because my husband said nobody would ever say that I was a bit nervous but I really uh I.

Really have got to say it tasted uh so good Dr Paul's your husband is going to have to take back what he said about no one liking it uh she checked her glucose ketones before she drank it her glucose was 72 her ketones were 3.2 20 hours into her fast and she was doing some work outside uh she went came back in about an hour.

Later her glucose was 63 and her ketones were 4.7 that is a boost uh she did also say how she has stocked up on keto combo because after uh December 30th I am kicked out of the club I can no longer sell that so if you want it it is through that bundle now because we only have a few left and we are trying to get rid of it if we run.

Out of the bundle we will substitute in the bundle some other products that we think are great and totally uh want to um give you what I think is the package for the best way to begin that starts with the education that starts with the fastest way instead of clicking through my lives which are long and I talk probably too much.

Um they the fastest simplest way that Joel could have totally avoided what he's doing to his system which as a physician I would say stop that I continue to tell him stop that stop that stop that and we're going to talk more about what's really going on some of with what his cholesterol is doing because it went.

Up yes it went up and if he had watched those videos he'd know do not do not check your cholesterol for the first several months of Keto I say six months because most people well they do what Joel's doing they screw it up they don't listen they don't read the book first they read the book after they've screwed it up after they started after they say.

Why don't I feel as good as the next guy I'm like slow it down you've got some issues and this is where uh the the world of healthcare is not a capture in a Flash uh how to do this but there is some complex um understanding of what is happening in the ketogenic diet all right so that brings me to.

Um our lesson tonight about cholesterol you've asked many people have asked about uh cholesterol I get lots of emails about cholesterol and in True Form I I love teaching about this but in order to teach you the depth of what you need to know about cholesterol it is not a two second video all right so I'm drinking my pucker up I.

Am beginning uh this lecture let's go over to uh these slides and um there we go all right so we are going to begin with what happens in that cholesterol panel um I practiced this this lecture a.

Little bit on my the folks that showed up at my uh my go-to here we go uh the folks that showed up to my uh uh support group this morning so they can fast forward through this a little bit but I I first want to start by saying when somebody says doc I'm gonna go check my cholesterol uh or I went to.

The doctor today and I checked my cholesterol they drew my blood and they checked my cholesterol that's actually not what they're doing that is not what they're doing they cholesterol is a fat and if the fat was actually in your blood if they were really measuring the cholesterol.

Uh because it was floating around in your blood that they would be working on a corpse blood is water is an aqueous solution it's water-based if you put fat into that blood it will cause a blood clot uh if you look at the most deadly version of that of having a fat in your blood it's when somebody breaks their femur.

And the bone marrow which is fat slips into that circulation and as soon as that fat touches their blood a a Cascade of clotting starts to happen throughout the body and even though that exact fat never reaches the heart the the signal to clot the blood spreads like wildfire and they often die so cholesterol is the shortened term.

That we use we use silly things like good cholesterol and bad cholesterol we're going to try to put a stop to that tonight a little bit it's a hard it's a hard thing to break the things that I like to show you is what we're really measuring are the particles in your blood that carry cholesterol and cholesterol it's super important.

We've talked about this several times it is absolutely one of the CR I mean every cell synthesizes cholesterol every cell uses cholesterol um if you're looking for if you really wanted to like measure cholesterol you would measure a whole bunch of cells gosh make them brain cells fatty cells that's where cholesterol really lives.

These part particles that carry cholesterol from the place we consume which was your gut and then your liver um they're called lipoproteins and the thing I want you to look at first is look around the outside of this lipoprotein this is a this one's a chylomicron that's the big the biggest one comes right out of your liver and.

Those blue uh circles all the way around the outside represent a water attracted part of this is the phospholipids and by golly are those phospholipids um important in protecting they are they love water and so the water soluble part is on the outside the fatty part is on the inside and as they float around the.

Body they link up to other areas that want this nutrients and they give it away we're going to cover this in a what happens after it gives a bunch of it away is it um it changes sizes and we change the name now we call it a vldl but you can see that uh it's a lot smaller now what I was trying to show.

You on that last slide to this slide is look at how much tinier it is several other things change within that uh that that that carrying particle that little ball of of goodies floating around your blood uh this actually does float around your blood when I go to check your cholesterol I'm checking this lipoprotein uh ildl is part of that and.

The ildl is just the last version with less of the triglycerides in it and then finally it gets to our bad boy our LDL but ldls do not come in one shape and size ldls have different ranges of how fluffy or how dense are they I just do want to say hello to nurse Cindy I have everybody saying hello to her on the on the channel always nice to see you uh.

But uh what when I am sure nurse Cindy has in her experience tried to explain this to patients I don't think you can do it without a nice little diagram so what we're going to talk about is did you know that this fluffy beautiful cholesterol these are both LDL cholesterol they're both known as the bad cholesterol but what we should be.

Saying is they're they're both lipoproteins they're the smallest the lowest density of lipoproteins and I'm going to explain exactly how important those are and what is the difference between the The Fluffy LDL and that bullet shaped LDL all right so this is a beautiful liver I hope all of you have a liver that looks.

Just like this and when you eat a bunch of fat let's say let's say you're Joel and you eat a bunch of fat and you don't hardly have any carbs and because he does oh my goodness as he following the rules he's following them so perfectly I need to say are you a human you are definitely following the rules perfectly.

Um but what comes out of Joel's liver is a big molecule called this chylomicron I also want you to take a tiny little notice that that number right there is a 48 this is April b48 the most important word in that though is April B and these are Kyle microns these are beautiful big fluffy uh um balls of of goodies and when you give.

The goodies away I don't know if you can see it in this slide but it delivers out the triglycerides coming out of that that little spherical um um carrying unit that lipoprotein and here's your here's your ldls this now becomes uh that was your vldl excuse me and if you remember uh Joel's vldls he.

Had nine before the ketogenic diet then he had 23 of them a month into the ketogenic diet uh these are his idls that wasn't on his report and that's often left off of the port it's okay because his LDL his bad cholesterol uh was 113 before the ketogenic diet and a month into it it's 241 like off the charts holy smokes he's gonna die uh.

He's not gonna die so what happens next is those LDL cholesterols are empty they've given away those particles have gone around they've delivered all the nutrients they can and now they need to be recycled your body is very conservative it will definitely use every single morsel that it can so as those ldls have kind of.

Really gotten rid of everything they needed to as long as they can be they can get back into the liver and in a healthy liver there is no restriction they can get right back in you now have uh them recycled and what comes out the other side is more of these big fluffy filled chylomicrons okay so here is uh the problem so I want.

You to notice what happened to that liver this liver is not pristine this liver looks a little gnarly it has been soaked and bathed in insulin and it's been soaked and bathed in insulin for a couple decades because we got a whole bunch we got a whole bunch of problems in there and when that insulin is uh too high in their body first of all their.

Blood sugars are don't get into the Dr BOS ratios where they have a five or six blood sugars don't or not in the 70s or 60s they're in the 80s 90s hundreds 110s 120s 130s up to 500 I've seen them um and they do not make ketones they do not make ketones so uh you can see there there is a they're getting rid of triglycerides there these little these.

Little things coming out of the of these spheres are the delivery of the the juiciest of fats that's your triglycerides um as you continue to give them away they now get smaller and more dense now you get this uh the or they get smaller and they are a low density not a high density high density means they're.

They're heavier they're more dense um these are low density they kind of are filled with more cholesterol but most importantly the insulin uh the high insulin does not allow them back into the liver very easily it just takes a long we call residency time which means those particles that were out for delivery are now staying in the.

Circulation too long their residency time is too long um and that's that's real the real where the real danger comes from so we're going to take this into a microscopic level where this over here is your liver and this is the Epi so yeah this is the epithelial lining that little skin.

Lining on the insides of your blood vessels that if you were look to look down this tunnel the the skin that you see lining a blood vessel is this endothelial lining this is the artery wall where I put that star and then the red area is where the blood is Flowing so as you look at what.

Is typical this is normal those LDL cholesterols those bad cholesterols this is a nothing Burger they float along and they go into the liver and they get recycled and the story is no problem the one thing I want to point out here is you see this gray line here this is a this is a glycocalyx that is found on.

The inside of your blood vessels it is not visible I mean it's visible if you it's like one of those it's like a very um like kind of beautiful uh little little fingers if you would that are the the glycocalyx it kind of floats in the in the in the fluid there and it is not really visible um it definitely is.

Um but but it's but it's there it's kind of hidden what happens when insulin is around is the first thing to be compromised was that little glycocalyx and the glycocalyx uh was a protective value much like the gut lining has that slime layer inside its gut and you don't really see it when you do a colonoscopy.

When people have a arterial catheterization and the doctors kind of in there looking around or trying to put a stent in you can't see this glycocalyx that was supposed to be there it's a protective lining that's like the extra caulking if you would that keeps out the stuff that shouldn't get out of there so the first thing that happens when the.

Insulin is elevated for many years is their glycocalyx is gone the second thing that happens is that their liver begins to change so I told you how quickly went from pristine liver to not so pristine liver well in a setting of a high insulin the reason it's high is because you have a bunch of excess sugar so the first layer that's.

Happened to this uh this liver is that it got a whole bunch of extra glycogen stories that's those little white balls they actually have little bitty sugars in them if you look closely the next layer of pathology that happens is that they're the fat those triglycerides are getting stuck in the liver and as those triglycerides stay.

There for a day ah okay that's not great but if you've had high insulin for as long as Joel has that high insulin now has done some not nasty things to the liver it's got it filled with the storage bin is high and because of that it now gets hard or serotic so you've now got this liver that is jam-packed filled with a whole bunch of fuel the.

Kind of fuel that you want to have uh in your um in your circulation for cells that are hungry but um you have it stuck in storage because actually the circulation is filled with things that's not being used correctly so let's go back over here and now you'll notice over here this this liver.

That liver is the gnarly liver so the kind that yeah um is not what you want this is high insulin and here comes that LDL cholesterol again it's in the blood it's in circulation and yes some of those at first do get in but look what happened to that one cholesterol it got outside the circulation uh well it where did it.

Go it went to a place it's not supposed to go it went to your arterial wall now remember uh this is your LDL cholesterol your low density lipoprotein and although it started out as your good fluffy one over time it's kept discarding its nutrients what it's it did its job and it changed the size it's it got small enough that it could wiggle.

Between those epithelial cells those skin cells that glycocalyx is gone it tucks into the arterial wall and now your LDL cholesterol LDL lipoprotein or your particle carrying around cholesterol is different and that's where we're going to look at even closer what happens on the other side of that skin cell so that little sparkly effect.

There is my magical way of saying this is when it got oxidized this is when it went from these nice pretty blue water philic um uh water loving uh uh um particles to particles that are well they're they're labeled differently and as soon as the body said yep that doesn't belong here this oxidized LDL is.

Is not in the right place let's try to fix it and so guess which team comes in to try and fix something in the wrong place that's your immune system one of your immune cells is a white blood cell called a macrophage and a macrophage is going to come in and Gobble up try to gobble up this dang cholesterol it's in the wrong spot why is it here what's it.

Doing here it doesn't belong here uh and as it gobbles up that uh that that fatty cholesterol there's a couple of mistakes that that white blood cells are making uh think of it as like the white blood cells poop out calcium now it's not exactly how it happens but it's a great imagery that the white blood cells come along they're going to gobble up these.

The cholesterol that doesn't belong here and the more it gobbles up the more fluffy it looked when it was inside the microscope so we call them foam cells and so it doesn't just happen to one particle of LDL it keeps collecting more and more particles fit between the little lining uh that is um that has those cells so you've got uh.

You know this little space had that opening and then the white blood cells came in and then they gobbled it up and unfortunately they left behind some debris called calcium so you can sometimes see it but these uh these white blood cells are trying so hard to protect from a mistake that's happening inside your body and that little.

Cholesterol is where heart attacks come from this this sequestration of these cholesterol that are not in the right spots they are in the wrong parking spots get the heck out of here uh what they look like inside your blood vessels is this uh see if I can make that go again there you go is this this is bad cholesterol.

That is truly bad cholesterol now not in just the LDL particle it is cholesterol that your white blood cell said get out of here get out of here get out of here and if you'll notice this little rip right there in the lining of that of the skin is where the cholesterol can come out into the circulation uh in especially in a way that.

Um happens just like what that patient whose femur broke and they had a fat emboli this is cholesterol that is touching the circulation kind of like a zit it just it takes that those white blood cells came in trying to clean up a problem just like it did with a zit and it causes inflammation and the inflammation collects and it.

Gets this core Center of Juicy gooey white blood cells trying to do their job trying to fix this problem just like it's trying to fix the problem that's in that in that lining of your artery and when it gets in under high pressure or when it gets in a time of turbulence or when the blood pressure got a little higher or when the the lining of that of.

The inside of your blood vessel got too thin it popped it popped yes and that's where the fat now touches the the cholesterol uh and that's really where we have a problem yeah so in this image here let's see I might want to switch this to a different scene let's see if I can make this happen.

Without anything going wrong yeah let's take my head and get a smaller and go up here all right so here are these two cholesterols so they're both um bad cholesterols one is your fluffy LDL cholesterol the other one is a bullet LDL cholesterol but in the world of cholesterol they're both called bad.

Cholesterol that's the terrible name for it they're actually low they're they're lipoproteins one has a purplish color and one has a bluish color but what's most important is that when you look at the impact that these cholesterols have this fluffy this this fluffy one right here is not associated with heart attacks uh this.

Bullet one does have the risk of heart attacks this one when you have increased saturated fat like what Joel just did it shoots up it goes up for sure it's now in circulation I'm it's now got a new level of um a new level of uh of like delivery route that so much coming in that it's almost overwhelming.

Uh when you look at how to make these bullet ldls you see those rise these bad boys get high with high insulin or in a place where when you're eating a high amount of processed carbs um this is the real sad part is that yes I can make your LDL cholesterols get smaller I mean that the numbers go lower.

I can take that lab test and make it look normal but my statins really are working at the drugs that are in or at the lipoproteins that are in circulation and when you look at the bullet ldls these small density ldls they are not improved by statins and therein lies the problem how in the world do we tell the difference between a.

A uh a fluffy LDL versus a um a bullet LDL several weeks ago you saw me do my labs and I went through all my labs and talked about ferritin and how important it was and your uric acid and um what else should I check I check A1C I checked a whole bunch of labs and I got to cholesterol and I said.

We're gonna do this a different time and then I accidentally flashed up one of them that I did not want to have on my slides that day you can go back and look at that but uh it's because I wanted to have this discussion to say well if I was just looking at my labs and knew nothing about the diet I was on my cholesterol looks High it looks like.

I should have a problem my even my bad cholesterol even looked high but you'd be missing the the uh the discussion about well I've been on keto for a while I'm not I I I've made the mistakes that Joel has made I've seen patients make the mistakes Joel's making but I'm not doing that right now I am in steady mode pretty.

Stable I screw it up sometimes but for the most part I got a plan um so let's take a closer look at what exactly I wanted you to get out of this discussion let's see if I can do it I think it looks easier for these are these are really nice slides let's just see if I can do it this way I might need.

To have my head down here okay so first things I want to point out is when I was showing you the differences between all of these particle sizes this chylomicron see how big it is it's like a thousand nanometers and these little bad boys are the particles that um that we're actually that's where the that's where the importance comes so yes.

Chylomicron is 100 to a thousand nanometers um when you look at the size of your um V ldls remember Joel's did go up vldls are on everybody's lab panel if you've got a cholesterol panel it's there um and 50 nanogram nanometers is how big these are so it's much smaller the part.

That I want you to notice is this yellow part is actually the cholesterol and if you if you reverse in the slides and go look that was the cholesterol also in that um in that chylomicron these are the triglycerides these little Tri Reds with three lines representing a little bit of glucose and.

Then three strings of fat this is the juicy stuff now there are other things inside these particles circling around delivering the fat but when you're giving away the triglycerides as fast as you are notice that the density or the amount the the concentration of this cholesterol these yellow ones um these ones is getting higher when.

They get smaller we're giving away triglycerides so those little um the the concentration of or the ratio between cholesterol and triglycerides is getting um more closer to one to one yeah like at IDL you can say oh look at that we got about as many triglycerides as we do have.

Um um the the cholesterol components and that's not an accident that's that's what that looks like but here is your bad cholesterol here's your LDL cholesterol notice that the size of this is between 18 and 28 nanometers in fact the key number to look at when we're going to try to.

Decide Do you have a do you have a good bad cholesterol or a bad bad cholesterol and the answer lies within well what's it let's start with what's the size of that cholesterol uh so a fluffy a fluffy uh bad LDL particle is at least 21 nanometers or bigger um if you get closer to 18 and again.

This is three nanometers smaller uh that 18 nanometer is where it now can fit between those epithelial cells and as you look at how that changes over time you'll see that not only does the size change but it's there are several other components that end up on that.

Cholesterol on that that carrying unit for cholesterol that particle so the first thing that I want you to look at is when you look at the chylomicron and this is your vldl IDL and LDL okay the one thing oh hey hold on here the one thing that all of these.

Have in common is uh they have oh I didn't Circle they have an apob they have an April B I have an April B okay that's very important we're going to talk about that in just a minute uh that when you look at the amount of time The Residency time that your LDL cholesterols spend in circulation these.

Two have the same amount of of actual cholesterol those yellow things the same number in both of them uh this one has a this fluffy one has a few less of those triglycerides those those red squares um excuse me this one has a few less of them because it's been in circulation longer it's been you know dishing them out to other particles in your body it's.

Hanging out it's trying to do its job it can't get back into the liver uh when we have a when we give you a Statin a Statin whether to know what they do the Statin it has a receptor uh that it impacts when it goes to say I want to take you and put back into the liver it's going to use this receptor.

And we know that receptors have a let's say a half-life of two days if I give you a Statin the half-life of that receptor lasts longer it is two to three times as long so I have more little fellas gobbling up and putting them back into the liver recycling them back into the liver unfortunately these oxidized ones have.

Two things that happen to them number one uh they don't come near the liver as much they end up in the wall they're small so they end up in the side of uh the side storage pocket which is in that arterial wall and so they do not get recycled back up with what a Statin is trying to do these bad boys do I mean good boys do.

The other thing that a Statin does is it stops the synthesis or the making of cholesterol and so again that's focusing on those little fellows inside there uh it's a minor effect is on the synthesis of that cholesterol the the biggest impact that a Statin has is it helps it you know gobble it up from the blood and.

Put it back in the liver and although that technically is a very good thing to do um it would be way better if you just decrease the insulin if you've decreased your insulin to allow that but that's that's for a different lecture we're still trying to figure out why is it uh.

That well we're going to go back to that one sorry I got to remove the okay I need you to do this I'll hold on um oh there that's what I wanted to do I wanted to clear it so the the other thing that happens so the first thing.

That happens uh was that this oh shoot I don't want to do that how do I get that to go away um oh it's not liking that though now now I can do it I'm really getting technical okay trying again all right so this uh these two LDL cholesterols uh the first thing this.

Purple one did was it was exposed to insulin uh it was exposed to those that high inflammatory State and over time when it um got smaller started slipping into the side wall as a way to protect it that that's one thing that made it very deadly but the second thing that happened is it starts to collect what I.

Like to think of as a danger tags and these danger Tags I'm going to draw them as spikes on here and if I could um if I could I'd give them a name the name of those spikes would be l P little a now in the chat I want you to tell me if you know what your total cholesterol is.

And your your bad cholesterol you don't have to tell me the numbers you can't it helps me it's lovely to see those but let's go back to the chat so everybody can see this while I uh propose something because I've been talking for a long time and I I really do care about uh teaching you what uh is in store for folks with.

Um trying to figure out well doc which cholesterol do I have do I have the cholesterol that's light and fluffy I know with pretty good certainty just based on Joel's numbers that he has a whole bunch more cholesterol floating around and his Labs confirmed that in fact uh he even had an A1C done and his A1C went from 4.6 really good number to.

5.2 why did that go the wrong direction yeah as a discussion for another day too what Joel didn't have was a couple of numbers that would have actually predicted um well what's the size of that that cholesterol and what is what are the numbers out there that don't cost a million dollars that do need to be.

Checked by patients to say your risk of heart disease is highly linked to these two labs and as you can see we're 45 minutes into this lecture and uh you you know a lot more about cholesterol but part two is going to be next week when I tell you that between now and next week I want you to call your doctor or many of the.

Labs if Quest and LabCorp you can order these on your own you can say I have an account I would like to order my two Labs on your own and they are not your cholesterol panel the two Labs that I want you to order let's go back to um here and I go back to here.

This way the two Labs that I want you to order is the a PO B and your l p l a okay if you remember from those slides and you can reverse back and look at them the tags on those cholesterol particles those little units that are delivering that are circulating your blood that are there to deliver this.

Beautiful nutrient that every cell in your body needs called uh cholesterol they it has a flag on it it has a marker on it called APO B APO B is very important for telling me the risk of how How likely is it that you're going to have a heart attack but it is super important to know this number and this number in fact if I had.

If I had to pick one number to say holy Hannah if this number is not normal in you um you you have to go see somebody for that you cannot depend on a YouTube channel that's teaching you about cholesterol in a way that I hope is transmitting to your brains uh LP little a incredibly astrogenic incredibly.

Linked to the um the aortic valve actually stenosine uh it's it's it's it's a good guy it just has a really bad rap and you want to have him just in tiny little doses not in high numbers and as you're a little as your your lipoproteins got smaller and smaller and then they waited in line and waited in line to get.

Recycled into your liver they not only got oxidized they then had this nasty little tag put on it LP little a and if you have a high number of LP valet that's what we're going to learn about next week okay provided I get all the labs the slides done those slides took us a long time you need to say thank you to jack for all the hard work you put.

Into that uh we are uh going to uh go over to your questions because you've been writing in some good ones here I have the gal the the folks my my magical elves Angela Michelle in the background helping me uh pick the questions that match for tonight's video I do want to say a couple of announcements that I did um I am speaking at low carb USA it is.

Early in January there is an addiction component to this which I actually didn't know about when he first asked me to speak uh and so that's happening on Thursday I will be there for that and I've just been asked to speak uh give two like two lectures while I'm there so the first lecture it was is on my updated version of why your average.

Blood sugar matters most I've put some really good uh evidence-based slides in there about what happens to that um and but the second one is a lecture on addiction and what that has to do with the ketogenic diet so I I'm super excited to be part of that if you cannot make it it's one of the kind of.

Conferences that I would highly recommend that you support by giving them a uh online buying the online version of it it's obviously cheaper but it also they're a really good really good organization that puts out great content and for the medical providers out there really trying to learn this kind of.

Stuff this kind of level of Education um I would uh there's not a lot of them that give continuing medical education points and this organization does now it's a lot more work for us to to do the paperwork but it's definitely the kind of commitment you want from your speakers so.

Here's my shout out somebody actually just asked will Medicare pay for April B it's 12 just order it don't uh yes Medicare will cover it but you got to go to your doctor then you got to talk them into it and you gotta it's 12 bucks it's cheap just order it um okay let me go to your questions here the other uh news that I I reached out.

To keto candidate ask if they have openings in their speaker um their speaker profile and I was afraid that wrestling season was going to get in the way of it but it totally doesn't so I'm I'm signing up for speaking at ketocon too so Dr baz we would love to hear your thoughts on how Vitamin D3.

Plus K2 affects the arterial plaques yeah again uh the the key components here are that vitamin D um uh there are not very many supplemental vitamins that I recommend I think a really good diet does do a pretty good job with the exception of k2d3 and iron but I do iron in the form of.

Liver the reason why I really support uh looking at Vitamin D is you can measure it when you're on an intervention for your health and you do not have a way to measure the metrics of what you're doing that's where I get confused the patient gets confused they say Doc I've done everything I'm doing everything and I'm like.

Let's look again I've I've seen a few of you uh measuring and measure it you don't need to tell me you need to tell you when it comes to vitamin D3 K2 the vitamin D component is um again the most powerful part about vitamin D is what it does to the inside the nucleus it is a fat soluble vitamin that once it is inside the cell the.

Mitochondria gives it a little wrench it gives it a little twist which allows it into the nucleus once inside the nucleus every single cell makes a different set of proteins it transcribes remember that RNA DNA transcription codeines guanine a to t g to C those base pairs that you're doing that transcribing of proteins yeah that's what happens inside your nucleus.

And vitamin D stimulates the production of specific proteins for every cell those vitamin D receptors are on the cell they get transcribed they get changed inside with the mitochondria and then they get transcribed into different proteins the white blood cells make certain ones heart cells make certain ones brain.

Cells make certain ones when you look at uh how many different things vitamin D can do it looks like snake oil like how can it do so much it's because it is a hormone it is a fat soluble hormone so in my keto patients in my insulin resistant patients in fact in Joel his Vitamin D is in the 30s that's too low it needs to be 50 or.

Greater I need his heart transcribing certain proteins that help it function better I need his brain transcribing certain proteins that function better and when you look at the calcified plaques there's another fat soluble vitamin called K2 and K2 helps to regulate that the calcium doesn't just get shuffled into your bones or Worse.

Excuse me doesn't just get shuffled into your arteries it gets uh it gets sent to the places in the body like your bones like the kidney that needs to use it like the different places where calcium is a coenzyme where it augments the way enzymes work cofactor for enzymes that's what I meant to say so the use of both vitamin D and K2 they're fat soluble fat.

Soluble doesn't mean that oh you should eat more fat no it's it is dictated it is it is under the dictatorship of insulin just like every fat molecule like your cholesterol this whole story about why in the world is cholesterol going why are those LDL particles ending up in that arterial wall and it is because insulin is attacking that.

Process of recycling things through your uh through the liver like it's supposed to be done properly um all right so I need uh so the next question says is that an average size so whoever copied and pasted that one in there if you can tell me which what size they're talking about there size of I I don't know what that size is.

Um all right so let's go back to the questions here and it says so taking high dose of niacin to lower cholesterol raise my HDL okay so is taking high dose of niacin to lower cholesterol bad uh I would not recommend that actually now there's been several schools to this in the past so it's a great question uh.

It is got a lot of if you think that biochemistry for cholesterol is bad is complicated wait to see what niacin does niacin has several layers of um of how it impacts not only um the blood vessels and how they vasodilate causing a flushing reaction but also the inflammatory processes inside the liver inside.

Those arterial walls and oh my goodness um the the particle sizes how particles are are regulated uh definitely alter the Improvement of or the the making it more fluffy and making it less the punch line was niacin although it will shift some numbers it does not lower the lp lilay it does not lower the April B and those are the numbers that.

Matter those are the numbers where you say I'm I'm tired of telling people you can't just look at bad cholesterol and then to get them to this part I mean I used pictures in animation and it took me an hour and I wonder if I lost my audience or if you understood um and we didn't even get to the part where okay so now the next level level.

Of that is um is what we're going to talk about next week so I think I see uh let's see let's go to the next question we're going to do at least a couple more questions because they're really good questions um Charles writes in and says Dr baz um.

Why shouldn't someone who was on statins for 10 years plus not get a coronary artery calcium scan all right so when you look at what the Statin has done gosh this is so hard to say because it's like a Trail I've written for statins I there are still places where a Statin is the.

Right answer I know that's going to make me the Antichrist to some of you but there truly are um when you look at coronary artery calcium scores as you watch the impact of which cholesterol was getting upregulated was you know circulating back into the liver recycling if you would it was those.

Fluffy cholesterols that that had a nothing Burger they didn't they weren't oxidized they sure as heck didn't have those red spiky things on them anymore um and they ended up in that arterial wall and that that macrophage then came in and it tried to solidify it it tried to push it away from the lining so that it.

It wouldn't erupt like a pimple but as it did that it left behind a bunch of calcium those white blood cells while they were gobbling up doing their job they pooped out some calcium again it's not exactly how it happens but it's great imagery to remember why is there calcium in the artery wall well because that oxidized calcium got in there.

Because the glycocalyx wasn't there anymore and your LDL cholesterol just kept getting smaller too small and fit through the the place where those skin cells just plop through so if you're looking for um a coronary artery calcium score which is important.

But you've been on the Statin for 10 years you're not going to give me any the information just says yep he's been on Statin for 10 years so I I wouldn't I wouldn't do that I wouldn't spend your money because you're going to confuse you and the doctor and it doesn't help you.

Uh there are things you can do to help yourself like get your A1C lower okay so is this the cause of atherosclerosis yes the cause of atherosclerosis is that the particle that carries cholesterol called LDL particle got too small fit between the epithelial cells got oxidized and then these spiky things got onto it and that.

Is what causes the heart attack um I think I may be eating too much protein although I'm eating lots of fat how do I know if too much too much protein I am only part way through the keto Continuum books the protein part becomes a very Lively discussion for people who um so I had this question actually today.

On my it was day two of the brains course and I let folks ask me whatever question they want when it gets to be their turn and so uh some questions about brains but some questions about well how do I know if I'm eating too much protein and this gal was almost 70 and so I do not want her to lose any protein I want I mean any muscle mass I.

Do not want that but um I tell people don't focus on too much protein it's really hard to eat too much protein what you should be focusing on is what are your doctor boss ratios especially in the morning and right before you break a fast how well does your body.

Um handle stress and one of those stresses is fasting we had lots of discussions today about when people break their fast and how they get diarrhea and that um you know it's not it's not nothing it's actually a sign that that stress caused you to have a reaction that you shouldn't have had it should not have.

Caused you to have and everybody has a little loose stools when they break their fast but um The Diarrhea for two days and that's feeling awful after a fast that says whatever stress you just did was too much uh so it brings you back to saying too much protein is where people focus as they're saying hey isn't this causing.

My insulin to go up no no stop don't do that look at your numbers look at how your body is responding what is your glucose what is your ketones and those two things are run by insulin and notice I'm not telling you to measure your insulin insulin does this volatile thing bounces all over the place if you have constipation on the.

Way to the lab you're going to screw up your insulin and I'm not going to know is it truth is it not truth is it high because of this did you get no just check the two things the insulin controls moment to moment which is glucose and ketones speaking of which let's check my glucose and ketones.

I will tell you that I have not been perfect since the 21 Day course that 21 day metabolic boost was awesome for me I cleaned up a couple of habits that had slipped back into my life um and that I kept waiting for a good excuse to fix them and that was a perfect habit and I many of them have stayed some of the habits have stayed.

Away but other habits have not stayed away I've had to see them sneak back in in fact the last 72 hour fast I did was totally during that 21 day boost so I cannot wait to do it again in September in um in February because I'll clean up a couple of those habits that have snuck back in but I.

Will also work on a couple of things that I kept telling myself I need a good excuse to to focus on that so yeah ketones 1.6 glucose 57 and again that is um that's pretty good for me I pretty hyped up on this topic so I'm probably burning some pretty good fuel tool again I don't check my numbers because I think.

It's like nothing I check my numbers because yeah um I needed I need accountability too I know that folks are going to be watching how how did I do on my fast I started on Sunday I started a little later than I should have and then I was definitely crabby last night I went to bed because.

I was trying not to be mean to the whole world because I haven't wanted food and today was pretty good I had a couple of slumps in my energy but and I really wanted to eat but I kept thinking about what I'm gonna do on my drive home and so somebody asked me when do I break my fast and in a perfect world I wouldn't break them until tomorrow.

But I have had this habit which is I have these Crumbs from carnivore crisps and the only two things in this are meat and salt and if I was looking for a stocking stuffer you can buy a whole bunch of these crumbs it's like the best antidote for my fast um it's a portion controlled uh I'm I.

Like them I even add a little extra salt but I show you that to say yeah I I get to this part and I'm done fasting and I want to eat and although sardines would be lovely I think I've only had two cans since uh since the 21 Day Challenge and I mean I think I had 15 of them during the 21 Day.

I mean preparing for teaching the coaches so we are looking to do that again uh in early January we'll talk more about that keep in mind that if you want to learn more about how cholesterol is processed uh the end screen on this is going to have a video that talks about what happens with processing cholesterol it's a lot shorter it is.

Some of the same animation with just a little bit different Twist on it and I I do want to hear what you think of not have any commercials during my live is that helpful for you I'm super thankful for the people that show up every week and I look forward to finishing this lesson next week I will say Merry Christmas and good luck with.

Uh getting your hands on one of those Christmas bundles I think it would make a perfect gift for somebody who wants some help with the keto diet not somebody that you think needs

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