Cholesterol Basics 101 by Dave Feldman & Dr. Berg

Cholesterol Basics 101 by Dave Feldman & Dr. Berg

Cholesterol Basics 101 by Dave Feldman & Dr. Berg

New To Keto But Want To Grow Your Knowledge?

More specifically, you want help with Cholesterol Basics 101 by Dave Feldman & Dr. Berg?

hello everyone so before Dave Feldman does his talking cholesterol I wanted to first give you some basic understanding of the definitions he’s going to use so you really can digest and understand everything that he’s going to say okay so we’re gonna start with this definition of cholesterol so the first thing you need to know is that cholesterol is not really a fat it’s not a fat it’s a fad like waxy substance okay so let’s start with the purpose of cholesterol number one it provides the raw material to certain hormones okay number two it makes up all of your cellular membranes so in other words the outer shell of your cells are made up of cholesterol 3 it makes up part of the brain without cholesterol the brain will not work correctly also a part of the nervous system the insulation around the nerves is made from cholesterol 4 it helps make bile so what’s bile bile is the detergent that helps you break down certain fats to turn them into fat type of vitamins essential fats that you need like omega-3 fatty acids and it helps you make certain vitamins like vitamin D so without cholesterol you can’t make bile and you cannot make vitamin D so it has a very important purpose everyone’s focused on cholesterol being bad it’s gonna clog your arteries but cholesterol is very very important to actually help make up part of the body tissues alright so the next thing you need to know is that the body makes a lot of cholesterol itself it makes roughly about 3,000 milligrams of cholesterol every single day that encompasses 75% of the cholesterol in your body so if we were to take you to cluster out of your body 75% of it would be made by your body and only 25% of it comes from the diet the next point is that cholesterol is a part of plaquing as in a clogged artery ok but it’s not the cause of the plaquing it’s a part of the crime scene but it’s not the criminal just because there’s cholesterol in a plaque doesn’t mean that it caused the plaque cholesterol is there as part of a band-aid trying to heal something as you’re gonna find out it has several really important purposes and let me just touch on this word triglycerides okay simply it is a type of fat that makes up your fat cell it also can be consumed from your diet to supply energy or be used to store fat carbohydrates that you eat can be converted to triglyceride in the fat cell so triglycerides are called lipids and what you need to know is that cholesterol triglycerides and other flip it’s are hydrophobic which means they don’t mix well with water okay like oil and water do not mix so then the body has to do certain things to make it mix with water to allow the fat to travel to the rest of the cells why because it provides the raw material to hormones and helps make up the cell membrane help support the brain makes the bile of vitamin D how is it going to do that if it can’t be transported so your body adds a protein to the lipid and that is called Lippo proteins okay let’s go to the next section on that okay so in this section we’re going to talk about the five lipoproteins okay lipoproteins basically our boats or carriers to transport fat and cholesterol to the body because fat doesn’t mix well it’s hydrophobic with water so we need to add a protein to the fat to allow it to travel through the body okay so the first one we’re gonna talk about is this thing called a Kyllo micron okay what is that basically that is a boat that transports the dietary fat that fat from your diet from the small intestine to the cells okay so this is created when you’re eating so it’s a boat that takes the diet fat and it brings into the cells okay another name for this chylomicron is ultra low density lipoprotein so let me explain this because it’s a little confusing ultra low density because if you look at the other ones in relationship to this one you have something called very low density lipoproteins intermediate density lipoproteins low-density lipoproteins and high-density lipoproteins so if something is more dense high-density that means it’s more compacted okay so you see how small it is now look at a chylomicron it’s ultra low density so it’s bigger it’s not very dense it’s an ultra low density particle okay so then we have very low density lipoprotein it’s smaller and it gets smaller and smaller and smaller and reality we’re just making something more dense so that’s just another way to understand the relationship between all of these to try to make it a little bit simpler okay so the chylomicron is created when you’re eating but when you’re not eating and you’re fasting okay what we have to do is we have to take the energy or fat from the fat cell and transport that to the cells okay we need a shuttle for that and the first shuttle is the very low-density lipoprotein so basically you have this boat that has this energy that it’s going to start feeding the cells okay feeding it fat triglycerides and then it’s going to end up as a low-density lipoprotein so you can kind of look at this is these three together as a kind of a transition phase so we’re going to go from here to here to here and by the way there are other functions than just feeding the cell as well and number one it’s going to provide vitamin E and other antioxidants to actually neutralize free radicals help neutralize pathogens there’s even a little bit of carotenoid in here which is another antioxidant so the very low-density lipoprotein carries the fat from the liver to the cells and then this would be the next phase with the LDL which when it’s done with its purpose it gets recycled by the liver now what is the HDL the high density lipoprotein do it carries excessive cholesterol that’s still lingering around from these guys back to the liver to be recycled or used for some other purpose so each of these boats their own percentages of triglycerides cholesterol and protein okay and that’s what makes them different all right so now that you have a little background let’s go right into Dave Feldman’s talk he’s a senior software engineer business developer and an entrepreneur in April of 2015 he began a low-carb high-fat diet and after experiencing a significant rise in his total cholesterol committed himself to learning everything he could about cholesterol and the lipid system lipid his fat right using his engineering background so here to make a pretty complicated subject simple for you are you gonna come up here and say something Dave Felton thank you thank you so much I’m curious do you guys do you guys like plays yeah yeah I mean you ever go out to the theater kind of get to enjoy a night on the town well what I’ve liked about this conference is fairly early on when Eric approached me we started chatting up this idea that would sound outlandish to other conferences but this isn’t like other conferences right now so I would like to introduce to you an important cast that’s actually at play right now inside your body please welcome the cast of the lipid system [Applause] we’ll stop yeah just stay here come on I know I know we really decked it out on the pricing here now I’m gonna do is have you guys a little bit more over here and spread out so that the podiums not too much in the way hope you guys have a decent view nabiollah ford here perfect alright so how many of you guys had a fatty meal Wow a lot of you a lot of you had a fatty meal it’s interesting well what happened to those fatty acids that were being pulled through your digestive system well they got packaged into something called a kite sorry a triglyceride now the triglyceride as we see here by your digestive tract is having trouble getting it into the bloodstream and that’s because it’s hydro phobic but she has about to transport and that is called the chylomicron a lipoprotein now the chylomicron is kind of like a waiter it’s moving around your bloodstream going to these different cells and oh one of the cells would like a triglyceride and now that all of the triglycerides are off of the chylomicron it then gets absorbed by the liver however you’re not always eating right so at a certain point you are fasting you hear much about fasting at this conference your body should be in a state of fasting not just overnight but also between meals so then what happens don’t we still have some cells that need energy you can tell that’s by the way the signal that you know the cells need the energy well fortunately the liver can help out and it does so using the fat cells the liver has its own boat known as a VLDL and there it is with fat cells ultimately supplying those triglycerides through the liver to the boat and now it does the same thing that chylomicrons were doing but from its own boat go ahead and now that it’s relieved of all of its triglycerides unlike the chylomicron it has a second job it is an LDL particle so now it has things you hardly ever hear about one of those for example is that it actually helps to neutralize pathogens it does so with something known as vitamin E ooh was a nice ring but now that it’s totally out of vitamin E what can it do with this last pathogen that’s coming forward well fortunately it can bind to that pathogen to help clear it and that ladies and gentlemen is your lipid system at work as we see it now thank you so much oh my gosh we were gonna do a a theater bow here we go okay get one more big round of applause for everybody you volunteered for this thank you guys so much so now if I could get my presentation up I think we’ll be ready to get started hopefully this isn’t double making me all right keto and cholesterol so it’s kind of a sentiment that I’ve seen the entire time I’ve been working in this and it’s will be actually three years next month but very early on in the literature I saw effectively what you see in this cartoon which if you can’t read it says we already know the killer is cholesterol we just need to get enough evidence to prove it and that’s effectively what I’ve seen over and over again and again I’m an engineer like Iver you can’t say something like hey we’re at risk for a meteor strike and it’s incumbent on me to prove you wrong it’s up to you to actually prove the meteor strike is in fact imminent that we should do something about it so let’s go back before three years ago in my own health journey this was what I would call my fighting weight and I don’t know if any of you guys have ever done this I’ve done this when I was running and yet I still somehow had a gut if I knew I was coming up on a camera while running I try to suck in my gut I try to get the shot right well I certainly may not be able to see my gut in these shots but you can certainly see my neck in my face I was definitely a lot puffier this next picture was actually my engagement photos with my wife and at that time I weighed quite a bit more than I do now well this is me now start Quito in April 2015 lost 25 pounds and the big moment again was about three years ago next month I became obsessed with cholesterol research since seeing my total and LDL cholesterol skyrocket and after that I began extensive in tawan experiments to learn more and for those of you in the room who know what I mean by extensive in of one experiments you know the inside joke for those of you don’t let’s have a little journey together so I take metrics constantly this is back these are old pictures you can tell because I have a precision extra when in fact I like to use keto mojo all the time now I have it for both glucose for ketones I do blood pressure I do like a whole bunch of metrics but this is probably what I’m most known for I take a picture of every single thing that I ingest everything in fact at every conference I make everyone aware of this I will give you $100 if you can capture me ingesting anything that I didn’t already take a picture of first but I’m gonna warn you my wife hasn’t collected on that so it’s not looking good yes this is literally everything you may have noticed my left hand appears in it and that’s so that there’s always a reference for volume so if I’m not actually weighing it I can be sure that I know approximately what the quantity is but a lot of times I weigh it as well like you see I’m doing here with the shredded cheese and when I say everything I’m not talking just foodstuffs I’m talking also supplements and yes even water every single sip of water that I’ve had I even have a symbol by the way for when I go to a water fountain and I hold up how many fingers for how many gulps that I’ve done now why am I doing this why am i capturing every single thing I’m eating this obsessively well they have to match with my blood tests how many blood tests have I had I’ve had about a hundred and five in the last 35 months since my research has begun and the reason I emphasize this is because I take the data that I’m collecting very seriously to the extent to where when I’m doing these experiments I can show you just how tightly controlled it is that’s what’s going to bring me about to the inversion pattern now for those of you are not familiar with my work this will be quite amazing because this is really the model at the end of the day as to why it is that it relates back to me as a software engineer I saw early on that this looked like a network but these different things talk to each other and that’s part of why I was happy to put that play on just now was because you can’t look at the lipid system in a static manner you have to look at it in a dynamic manner and once you do and particularly the more you can kind of understand networks and software engineering in my opinion the more you can get why it can be the way that does and how well it can function so about I want to say two and a half years ago I was at low-carb ville and I brought these eight data points that was back when I had only eight blood tests and I took them over three months and as you could probably see by that dotted orange line it seems to reflect what’s going on above it in that sort of solid blue line that dotted orange line is a three-day average of my dietary fat that solid blue line that’s my LDL cholesterol so I took that to low-carb Bale and I stocked all of the different doctors there the different researchers and so forth but I actually happen to have a great conversation with Ivor Cummins as it happens to be the one other major engineer there and sure enough I said you know what I’m gonna do this I once I get home because this was taken over three months and it was about every like 13 14 days I’m gonna actually do one full week where I actually eat to a meal plan like you see here so that I can induce a curve to see if the LDL cholesterol will even follow it when I’m doing it one day after the next actually was going into the phlebotomist getting my blood drawn every single day so what happened that happen sure enough we see the reflection shown and then of course for my next one I said well heck what if I just go gin the fat what if I have tons and tons of it well we then see the lowest LDL see that I got since I started since then you can see with 29 data points the very beginning of my research what is clearly what I would call the inversion pattern and I’m gonna flip the axes on the left side flip it so that you can actually see how well it retrofits when you can see the inversion together unquestionably one of the most amazing things that I’ve ever seen and it’s amazing to me even more so that I’m actually just looking at nutrition labels coming to the macros and from the macros ultimately being able to predict accurately what the cholesterol levels will be in my blood now for those of you who are actually rendered the statistics mumbo-jumbo the Pearson and the regression those are amazing scores and this is what I call the inversion pattern the dietary fat inversion for LDL seen it’s really quite simple I’m right now recording this on a Saturday so if I were to have taken my blood this morning I would look backwards three days the Friday yesterday and the Thursday before that and then the Wednesday before that and from those three days I feel that I can make it fairly strong guess as to what my resulting LDL cholesterol would be now how is it that I can get to that well I’m going to explain the model to you and I’m going to warn you this may be one of the more technical areas but for the mumbo-jumbo slides don’t worry I’m gonna slow down and walk you through it now this is one of those slides the truth is this is what you hear about all the time in the ketogenic community and even outside the ketogenic community that carbs and protein can be converted to glucose okay that’s great you also hear all the time about how fat can be converted to ketones and that’s true but here’s the thing you don’t often hear about it you don’t often hear about how fat can be delivered directly to cells until just a moment ago with our little play showing how they can get transported in these boats so these are the other pathways by which fatty acids can come directly to your cells but I want to focus on one particular one I want to focus on the one that includes that word I want you to remember there’s any word I want you to remember past this it’s triglycerides try as in three fatty acids three fatty acids they’re bundled up and they are basically the stored form of fat and they’re loaded onto these boats that your body makes and then sends around your bloodstream and I’m going to go off-script for just a sec does anyone know about what measurement just what total quantity of LDL particles you have in your blood stream right now is anyone want to guess no millions I mean you think millions how many think trillions obviously it’s a trick question it’s millions of trillions it’s known as a quintillion and it’s a one followed by 18 zeros your body makes these in large quantities every single week it actually will synthesize and then reabsorb these at very high quantities so it’s very keen to be sure that it delivers these triglycerides and ultimately then remodel back to LDL particles so putting it pretty simply if you were to think of a boat as a lipoprotein like this cruise ship what are the passengers passengers are triglycerides it’s right okay so what’s the cholesterol on this cholesterol is the life rafts what is the purpose of a cruise ship the cruise ships purpose is actually to mainly move around the passengers the cruise ships purpose is not mainly to move around the life rafts however you wouldn’t get on a cruise ship that didn’t have life rafts so you do still want the life rafts it’s still very relevant and you’ll see why a little bit more later so this is where I’m gonna kind of break it out into a little bit of a diagram and what’s neat is because of the play beforehand and everything else we’re helping to kind of set you up a little bit more so now let’s define what is an Apple lipoprotein okay so it’s an additional protein that binds to the lipid so you remember these little boats called the lipoprotein there’s five different ones that have different ratios of protein cholesterol triglycerides they’re carrying vitamin E as the antioxidant so the Apple lipoprotein is basically the captain of this ship that’s directing what’s occurring and we also have phospholipids which is another type of lipid that is used in making cellular membranes so now different apolipoproteins do different jobs and they have different names okay their name simply ABCDE h j l m and they have also subclasses of names so a would have a subclass of different names B could have a subclass so LD L uses Apple B HDL uses Apple a and IDL uses Apple II so you can see that these things right here have different effects for each one of these so what actually do they do well they have several things that they do so number one they would activate certain enzymes to help the conversion of a VLDL to an IDL to an LDL so basically they’re they’re making the fat dissolve from an enzyme called lipase and then I’m helping this unload so they can convert to this end product right here they’re also involved in transporting or directing the boats to through the circulation through the limp to the cells and they’re also involved in a communication of where they start to where they’re supposed to end up so very simply an apple lipoprotein is the captain of the boat or the ship and without them these lipoproteins would be a mindless bag of fat and cholesterol we talked about the chylomicrons they came from the small intestine they came from food you just ate they got loaded up with those triglycerides a nice big payload and then they got sent on their way to then go ahead and provide that to your tissues as soon as possible and once they did and they lost their cargo they then ultimately get absorbed back at the liver but there’s also a line that comes from the liver primarily of triglycerides that came ultimately from your fat stores from your body so when you’re fasting you absolutely are pulling more of those triglycerides into these vldls to then move around and they move into something known as ideals intermediate density lipoproteins which can also be absorbed and this is the story of energy delivery at least in terms of fatty acids direct delivery so now let’s talk about this other part of what lipoproteins do because it’s not just energy delivery their support you guys have all heard about HDL the so-called good cholesterol what they really mean is the cholesterol found inside these lipoproteins these boats and these are in fact very good they’re very good in your system you want them operating and doing very well you just like to see your HDL cholesterol very high well they also work closely with LDL particles the so called bad cholesterol but of course what they mean is the cholesterol found in low-density lipoproteins the boat now you notice this is the one line that’s pulling double-duty you see chylomicrons they’re pretty much delivering energy HDL is pretty much in a support role this is the energy delivery this is the support only only the purple line only vldls to ideals to LDLs have two jobs the first job being energy delivery the second job being in its support role and I showed you a couple of those support roles for example in being able to clear pathogens and also neutralizing free radicals with vitamin E also known as alpha tocopheryl you don’t need to remember that just remember the vitamin E so now you have to say okay now hold on I’ve heard that high v LDLs and triglycerides are associated with disease that’s true if they’re lingering how do you know if they’re lingering you know because it comes up in your blood test so oftentimes when I’m telling people about triglyceride so you’re being fueled by triglycerides they go and understand Dave I don’t understand I see that my triglycerides have dropped well that’s correct because your usage of triglycerides has gone up you’re now using your fat at a much higher degree and therefore there’s less and less triglycerides per boat and that might mean that there’s more LDL particles that started out as vldls that deliver the fatty acids you following me all right so now let’s talk about my low carb cholesterol challenge I’m gonna keep putting this into my presentations until it finally gets answered but I’m gonna say about eight months ago I open this challenge and I wanted to put it out to all sorts of different people I paint a lot of Lokar a lot of lipid-lowering experts people who are not necessarily low-carb who say look you guys are getting overly comforted when you have high LDL cholesterol just because you have high HDL cholesterol and low triglycerides I’m gonna constantly call this the triad those three together and I said that okay great so why don’t we start looking at the studies that actually examine all three of these together the high LDL cholesterol that so-called bad cholesterol the high HDL cholesterol it’s so-called good cholesterol the low triglycerides because from what I’m seeing doesn’t look as if there is as much of a problem but I want to see some conflicting data so I had this up eight months ago for six months while I had this up I got no studies they have done studies from the other direction again studies that have shown the opposite including from our friends at Framingham this actually was about four thousand different people and what I loved about this was it also excluded users of lipid-lowering therapy so you have a nice fresh batch of people who didn’t already have cardiovascular disease at baseline to see what a difference it would make for them with heart disease and this is what we saw I’m sorry is a little bit mumbo jumbo but the o.r above is called the odds ratio generally you want that to be as low as possible people everybody with an LDL cholesterol higher than 100 so long as they had low triglycerides in high HDL we’re doing fantastic well okay maybe this was including those people who had like say an LDL of 102 or 103 so fortunately on the same chart we also have people who have an LDL above 130 now that’s a lot higher but guess what it’s the same 0.7 now we’re talking significantly higher levels of LDL cholesterol it’s still so much better but I actually got another study this one I liked even more because in this case they were actually using men who of course are tend to be a lot higher risk for hardy vascular disease around 3,000 of them 53 to 74 and they divided the group by those who have an LDL of a hundred and seventy or less a hundred and seventy or less that’s extremely high or so we’re told to one hundred and seventy or more but if you look the darkened bar on the left side is HDL cholesterol that is above fifty seven triglycerides below ninety seven that’s the tertiary if the line is almost identical on both sides whether you had whether you had a high or low LDL cholesterol you ended up in better shape regardless so long as your HDL was high the difference is less than two thirds and 1% but I really want to call your attention to even more strikingly at the top end at the risk level where your HDL is low and your triglycerides are high that’s nearly identical regardless of your LDL so I can’t help it I keep looking at data like this and I say low LDL high LDL does it seem to matter as much as HDL and triglycerides now through a little more humor on it I said I had that challenge up for six months two months ago as of two days coming I went ahead and put some money behind it so I actually I actually I put it out there that I would like to give three hundred dollars to anybody who can find me at least one study just one study that showed those people who have high HDL and low triglycerides with high LDL would have a higher rate of cardiovascular disease and I even made it not even high I said just above average and I wanted to use the numbers that the American Heart Association a lot of different like heart.org for example just just just above average and I’ve yet to see one study and I’ve now put money behind it I have not spent one time of that bounty thus far so with that in mind I’m gonna show you some very very surprising data that came out of the most recent experiment that I did with the weight gain and this experiment before I start talking about it I want to emphasize to everybody don’t do this there are a number of experiments that I kind of have – okey half knots aid I’m doing this so that you don’t have to this experiment in particular some people get inspired to do experiments like I’ve done this is not one of them I actually knew I’d need to induce a state of hyperinsulinemia to accomplish this but I had an opportunity scenario you see I still had some weight left over from a previous couple of experiments my capstone in my sugar experiments you have to read up on my blog to hear about that I know how crazy that sounds and I had an upcoming trip with an understanding wife can I just set the scene there was one night where I walk into the bedroom and she’s reading and I go you know honey had really helped my research a lot if I could gain about 20 pounds of fat that’s how awesome she is so you know okay who are the experiment upsides obviously this was a chance to observe and compare cholesterol at higher weight on a standard American diet and to observe what happens when we change over to ketogenic diet almost all of that data will be available on the blog soon but there was an obvious experiment downside there is short and long-term risks I knew this going in and I made this clear to my wife and I made this clear to everybody else which is why I don’t want anyone else to do this obviously inducing a high level hyperinsulinemia is not recommended under any circumstance just avoid that in general if there’s anything you take from this conference it’s that so you can actually see this in the media that I was doing it around the time of the experiment you can see my face gets a lot puffy or my neck gets puffy or etc and this was the timeline is fairly straightforward Oh kind of cuts it off a little bit but you can see I was a little bit of a ramp up but it’s mainly through the month of April that I’m actually gaining this weight and here’s what I was eating bread a lot of bread I had a lot of cheesy bread I had a lot of sandwiches and I had that’s like a chunky steak meat pie that I was getting from a place called pie-faced in Australia and I actually intentionally avoided a lot of sugary treats because I just wanted to see you know what would come up with just a lot of starchy foods just just in case you were curious works like a charm in lots of weight I hadn’t yet gained my my goal weight of 205 so fortunately I was moving now into a new phase of the timeline which is for five days I’d be doing a controlled standard American diet experiment by that I mean I’d be eating to a particular plan what does that plan look like looks like this at 10 a.m. I’d have a footlong Subway sandwich at 3 p.m. I would have a medium pepperoni pizza and then at a p.m. I to have another footlong Subway sandwich total calories 4217 hungry do you think I gain weight I gained weight that didn’t quite give me to my 205 but I was I was really ready to stop it there by the way for anybody who’s contemplating leaving he doe going I wonder what it’s like to be like this I wonder if they’ve really enjoyed it you really honestly did not enjoy it I’m gonna say there’s only one thing I find that I miss when I ever I do these experiments that take me back to carbs and I’ll be honest about this it’s the hand-holding part that you don’t have to use like knives and forks as much that’s it that’s all anything else I feel like I get just fine with keto so okay I’d then take a huge number of tests and I want to point just to one in particular that third one from the left up there that’s my blood pressure it reads 140 over 86 I’m telling you my blood pressure normally is around I want to say 104 over 71 maybe so I really was ready to step off this experiment I was getting quite a bit concern now I then took a whole bunch of blood tests you only need to be you don’t need to pay attention to that but then I finally got to move to the keto phase which was a lot more refreshing now I got to have sausage scrambled eggs and I did have to keep it somewhat mundane so that I could control for all the variables so it otherwise was a lot of cheese and hard-boiled eggs with some almonds this was a lot better 2437 calories total carbs 20 and this is effectively what the controlled phase moving into the other control phase of the key to look like so I was literally eating the same thing in about the same way every single day and as I like to joke it’s kind of like Groundhog Day I like when the audience laughs that because I don’t feel as old when I say that and so now if we look at my triglycerides versus the way my triglycerides in the blood versus the way we can’t really tell a lot and that’s because this isn’t a relative comparison but what if I take my relative or if I take my weight and actually do a relative comparison so that we can see what’s happening between that five-pound Delta well we see that it looks like this striking my triglycerides were much higher during the standard American diet started dropping as I started dropping weight but here the kicker how many of you guys have heard show of hands how many guys have heard that your LDL cholesterol can go up as you’re losing weight well I’ve been hearing this for years and as the cholesterol guy I really wanted to know and now this was my one chance going from a standard American diet to keto and losing weight at the same time I finally got to find out and this is what that looks like my LDL cholesterol had started at a much lower level at 133 which I’m sure my doctor would have been a lot happier with and then as I started losing the weight it jumped up to 228 that’s a change of over a hundred milligrams per deciliter or close to a hundred milligrams per deciliter through a delta of just five pounds of weight loss so without question from now on whenever anybody is actively losing weight I say just don’t even look at your lipids but don’t even bother with it right this is what it looks like when we invert the pattern it’s actually quite striking and the pure scent is amazing it at point nine three six okay now I’ve got some new data you’ll notice this table over here is now starting to pay attention they were used to my other stuff so this is the resistance training experiment now enough you guys have heard but I was on a podcast recently otherwise I was the Peter Atia drive and I had a habit of doing this I have a habit of announcing my hypotheses in advance publicly I by the way I had a friend who said you shouldn’t do that because you’re gonna eventually embarrass yourself I said I thought that actually was what science was is too you know put forth your hypothesis before you test the experiment and he said and by the way he’s a PhD grad he goes well not anymore so I’m going to tell you them this is the mumbo jumbo and then I have a translation one for the next one so I’m gonna read this aloud again more Theory this is me talking to Peter I’m actually going to be testing intensive exercise myself in the next series of experiments that I’m doing there is a difference between those people who are doing things like say endurance running and those who are weightlifting or resistance training in that I think there is a greater overall gradient of reset Durr mediated endocytosis for muscle repair and growth don’t worry translation coming I could be wrong about that but I’ll be very curious to see if it turns out to be the case when I’m doing it myself and here’s the translation of what I really said I said I think when you make your muscles sore they eat LDL particles to repair and grow I hadn’t yet done this experiment at the time that I did the podcast so I was very excited to do it this ended up being the longest controlled experiment well before I get started let me tell you how I thought of that well you see this is a cell membrane and a cell membrane is made up of two things especially phospholipids it’s a kind of lipid and cholesterol that’s what that little orange guy is there right what’s a low-density lipoprotein made up of it’s made up of phospholipids and you get one guess Wester all okay so why is there this process that cells have halt that long word I used before receptor mediated endocytosis basically the engulfing of a lipoprotein why do they have that well i had speculated for some time that that’s because it’s for raw material certainly they would say cells have enough on hand to be able to repair and synthesize their own cholesterol for the most part I think that they’re right but what if the man climbs if you’re stuck in your home you may have enough in your garage to be able to do repairs to your drywall what if you don’t have enough wouldn’t it be great if Home Depot was driving by your house every five minutes saying aide you need anything doing anything so my theory was very simple I believed that if I could control for every other thing that there was that if I did some intensive exercise I would see a drop in my LDL cholesterol because there’d be a drop in my LDL particle count but in order to do that I would need to like live my life the same way day after day after day I think I could do that okay that was the plan try to keep everything identical from one day to the next for 20 days straight so I need to have a big washout period so I can be sure that I had a baseline and that was gonna have the same things the same way every day but on top of that also control for exercise I did my usual 2.5 miles of walking in the afternoon every time and this is what that looked like no these are not duplicate pictures this is literally the pictures they took on those days and at those times so I wanted to be sure I control for everything that I could including even sleep which I wasn’t always as successful with and this is what it looked like this was all of my lipid numbers taken for my cardio check day after day up until the first intervention period then this is what happened we see not only does it dip down but actually it starts to come back up right around the time I happen to be doing the second intervention phase and what did that look like that look like this you can actually see clearly very well delineated an intervention phase one we see a drop of around 11% on intervention phase two where I actually really hit it I did the weights I went to the gym and I also got vibration plates my LDL cholesterol dropped 13% keeping all other things the same now I can’t prove that that was entirely into cytosis I don’t even know for sure how much of that it was into scientists but do I believe that that’s still the case yes what does it take to do that it takes this takes being able to isolate every other variable this was also one other thing I just wanted to point out that I thought was fascinating as you can see on the intervention phase I was also doing glucose versus ketones the glucose are in the blue at the top the glucose actually didn’t change that much but the key talents sure did he tone production actually went up quite a bit or I should say perhaps the overage would have quite a bit because we can’t actually see the uptake coming into the cells I thought that was actually quite fascinating as you can see it starts to taper back down toward the end of the experiment now I told you about the fat experiment I actually want to come back to because one of the most powerful slides I have in any presentation I’m going to show you now and it’s really gonna drive home a lot of how I understand LDL cholesterol and why it is that I still have the state of uncertainty that I do as far as its risk so the carotid intima-media thickness test is a test along the carotid arteries that are along the side of your neck and it’s supposed to be effectively in his used in many studies as a proxy for a thorough sporadic burden what it’s trying to do is measure both the thickness of your intima and your media together now I have been tracking mine every six months starting in July of 2016 every six months to my surprise and appreciation my left carotid artery was slowly regressing not progressing it should actually be staying the same or going up with my age that’s the normal progression but yet it was actually going down my right carotid artery started much higher started at 600 I want to say around 680 something along those lines it actually dropped about a hundred and fifty nanometers while I was on this ketogenic diet but you remember I then after we go through all of this I then go ahead and have the standard American diet experiment I take my C IMT toward the end of that standard American diet experiment and I’ll say I was speculating there was a chance that might move north right well sure enough the left carotid artery jumped up higher than it had ever been before the right likewise in fact if you add that last line you can see just what a pronounced difference it is following four weeks of a standard American experiment standard American diet experiment I really have to say for any of you guys who right now are thinking you know what I think I’m gonna take a break from keto for the holidays I want this burned into your head now obviously I can be somewhat jovial about it because I am banking on the regression restarting again and if in fact that turns out to be true that’ll be some pretty powerful data there’s me making another public hypothesis about my data we’ll see how that turns out now this went from the lowest ever on both sides both sides not one side to the highest ever on both sides showing just how much of a difference it makes but this is the part that’s going to bring it home you see I already read in the literature several times one of the contributing factors not just to a thorough score attic plaque but in particular to the carotid intima-media thickness is as you can see highlighted a high lipoprotein levels high lipoprotein levels yet look closely if we add this last column on ldlc and LDL P I was averaging 200 LDL 200 milligrams per deciliter of LDL 200 or more throughout this period of time and my LDL particle count was 2,000 or more both considered well into the 90th percentile of risk at those levels my left and right carotid arteries were regressing so what happened to my LDL see in my LDL P I’ll give you a hint when I did the standard American diet as I hypothesized would happen my LDL see dropped to what it was before I started keto how much did it drop dropped to 130 my LDL particle was what would be considered a much more attractive 1130 so if you were looking at this chart alone and you were saying hey what’s the best thing that I could do to help regress the thickness of my intima you would come to the conclusion the thing I was doing in the first four rows makes more sense than the thing I did in the last row that’s why this data is so powerful even though it’s only ten data points so far I want to do one big thank you to my patrons I want to emphasize something to all of you I take no money from any corporate entity in order to maintain the integrity of my data and so I’m fortunate to have literally a hundred and sixty plus people who actually directly support my patreon which makes this research possible could you please give them a round of applause for me and thank you so much for your time I really appreciate it I’m really in love in this conference thank you so much you

This Post Was All About Cholesterol Basics 101 by Dave Feldman & Dr. Berg.
Cholesterol Basics 101 by Dave Feldman & Dr. Berg

Here’s The Video Description From YouTube

Take Dr. Berg’s Free Keto Mini-Course: or go here:

Download Keto Essentials

Dave Feldman and Dr. Eric Berg help explain the complexities of cholesterol, lipids, LDL, HDL as it relates to its true functions and purposes. Dave Feldman delivers this lecture at the Healthy Keto Summit. You will learn all about the basics and even see a skit put on by Dave to help you get the knowledge of this confusing topic.

When someone starts a ketogenic diet, they may have a temporary spike in cholesterol and LDL and if you don’t understand why this, you can be scared and stop the program. This is why this topic is very important.

Here are some basic definitions used in Dave Feldman talk during the keto health summit about cholesterol, and also its purpose.

Cholesterol is not a fat, but it is a fat-like waxy substance and the body makes 3,000mg (75%). It is a part of the plaquing but it is not the cause.

Triglyceride (are also called lipids) – it is a type of fat that makes up your fat cell.

Purpose of Cholesterol
• Provides raw materials to certain hormones
• Makes up all of the cellular membranes (the outer shell)
• Makes up brain and part of the nervous system
• Helps make bile and vitamin D

Lipoproteins – these are basically carriers that is used to transport fat and cholesterol through the body.

5 Types of Lipoproteins
• Chylomicron or (ultralow density lipoprotein) – carries the dietary fat from the intestine to the cells
• VLDL (very low density lipoprotein) – carries fat (from body) from liver to cells
• IDL (intermediate density lipoprotein) – Intermediate step
• LDL (low density lipoprotein) – carries fat / cholesterol from liver to cells
• HDL (high density lipoprotein) – carries excess cholesterol back to liver

Dr. Eric Berg DC Bio:
Dr. Berg, 52 years of age is a chiropractor who specializes in Healthy Ketosis & Intermittent Fasting. He is the author of The New Body Type Guide and other books published by KB Publishing. He has taught students nutrition as an adjunct professor at Howard University. He no longer practices, but focuses on health education through social media.

DR. BERG’S SHOP:

Follow us on FACEBOOK: fb.me/DrEricBerg

Send a Message to his team: m.me/DrEricBerg

ABOUT DR. BERG:

Disclaimer:
Dr. Eric Berg received his Doctor of Chiropractic degree from Palmer College of Chiropractic in 1988. His use of “doctor” or “Dr.” in relation to himself solely refers to that degree. Dr. Berg is a licensed chiropractor in Virginia, California, and Louisiana, but he no longer practices chiropractic in any state and does not see patients. This video is for general informational purposes only. It should not be used to self-diagnose and it is not a substitute for a medical exam, cure, treatment, diagnosis, and prescription or recommendation. It does not create a doctor-patient relationship between Dr. Berg and you. You should not make any change in your health regimen or diet before first consulting a physician and obtaining a medical exam, diagnosis, and recommendation. Always seek the advice of a physician or other qualified health provider with any questions you may have regarding a medical condition. The Health & Wellness, Dr. Berg Nutritionals and Dr. Eric Berg, D.C. are not liable or responsible for any advice, course of treatment, diagnosis or any other information, services or product you obtain through this video or site.
#keto #ketodiet #weightloss #ketosis

Thanks For Joining Us