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5 Benefits of Exercise at the END of Your Fasting Period

5 Benefits of Exercise at the END of Your Fasting Period

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5 Benefits of Exercise at the END of Your Fasting Period

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A lot of people who fast often workout at the beginning of their fast or after they’ve broken their fast and have eaten. This video, however, covers the 5 reasons why you might want to consider working out at the END of your fast instead. Enjoy, and I’ll see you in the comments!

References

https://link.springer.com/article/10.1007/s40279-016-0594-x
https://journals.physiology.org/doi/full/10.1152/ajpendo.1997.273.4.E768
https://biosignaling.biomedcentral.com/articles/10.1186/1478-811X-9-11
https://pubmed.ncbi.nlm.nih.gov/25316028/

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Does Exercise STOP the Effect of Fasting

Does Exercise STOP the Effect of Fasting

Does Exercise STOP the Effect of Fasting

New To Keto But Want To Grow Your Knowledge?

More specifically, you want help with Does Exercise STOP the Effect of Fasting?

there is a really cool balance between fasting and working out and I’m not talking about just like for cosmetic reasons like sure working out is gonna get you in shape fasting is gonna get you in shape now I’m talking about the cellular level like a really cool balance see it all comes down to two very specific things that i’m gonna talk about in this video mTOR and autophagy and right now that sounds complex but I promise it’s gonna make perfect sense in this video and you’re gonna have a clearer understanding of why balancing your workouts with fasting is the ultimate way to combine not only amazing metabolic and cosmetic benefits but also longevity and just long lasting health hey you are tuned into the Internet’s and leading performance fat loss keto and fasting channel new videos coming out every single Tuesday Friday and Sunday at 7 a.m. Pacific time but not just then we have videos coming out throughout the rest of the week as well I also want to make sure you head on over to highly calm so you can check out the premium performance apparel and I’m always wearing in my videos alright so what we want to look at today is we want to look at the balance between working out and fasting at the cellular level now to make some sense of this I have to give you a quick rundown of what mTOR is and what autophagy is okay some of you have heard about this stuff before so just bear with me if you have because I promise I’ll get to the good stuff mTOR stands for mammalian target of rapamycin an mTOR for all intents and purposes is just a very anabolic thing what mTOR is is something that is turned on when we are in an anabolic state meaning we have excess calories coming in and our body recognizes that as a time of excess or basically just being plentiful so it says great we have excess calories we can cell divide we can put up new cells we can have all this growth muscles can grow growth hormone can occur we’ve got i GF insulin-like growth factor all these things right it’s a time of excess okay then we have Auto Fuji which is the opposite Thapa g is a process where our cells are sort of condensing and recycling and trimming out the excess so we only have what’s truly needed okay but let’s focus on mTOR for just a second and then we’ll get into the autopsy side and how this all relates with working out and fasting so like I said mTOR occurs when your body is a rich ok I want you to think of it like that like times are good ok the stock market’s high the Dow is at 30,000 points everything’s good we’re spending money right ok this the same thing happened in your body your body’s like great I can create more energy I can create more adenosine triphosphate okay which is ATP which is energy it can grow new muscle grown ooh tissue all great okay but times of stress trigger low levels of emptor so basically when you’re stressed out when you’re literally under stress or when you’re not eating because you’re fasting your import levels go down but the opposite of mTOR is on top of G so when mTOR is down autofit G is high and when on top of G is low mTOR is high okay now again pay attention to that because it’s gonna make some sense as I get further into this video so at the end of the day we want everything cycled okay we want a cycle of high levels of mTOR and then low levels of mTOR okay high levels of em tour great because again we’re growing new cells it allows us to build more muscle unfortunately it also allows us to build more fat too but again gives us that time of excess for cells to grow Altaf G trims it down so think of it like this it’s a party M tours hi everything’s good top of G is the cleanup okay now on top of G cannot come in and clean up until the party’s done okay so I’ll top it you can’t come in and clean up all the cellular waste and everything like that until mTOR is gone until the party’s over so M tour is a party atop of G’s the cleanup crew now the cleanup crew can’t come in when the party’s still going on because nothing’s gonna happen so that’s what we have to remember is whenever we’re building muscle whenever we have an excess of calories or we’re working out we have mTOR activation we also have a higher level of waste and that’s just part of life okay so it’s like if you’re spending a lot of money or if you’re living rich you have a lot of waste – okay more money more problems right same kind of thing happening inside your body so let’s take a look really quick at the good and the bad of mTOR okay the good is increases in muscle increases in IGF where you can build more muscle increases in growth hormone okay increases in overall abundance inside your body an increase in mitochondrial metabolism the ability to actually create energy all good the bad potential for more disease extra waste downregulated immune function okay so when we have more improv and definitely can lead to increases in cancer and things like that because we’re increasing that cell growth but this is exactly where the balance comes in okay because levels that are too low with mTOR are actually bad as well so there’s a study that’s published in the journal Cell Metabolism it took a look at liver cells so hepatocytes okay and it found that when they were in a state of low mTOR for a long period of time they had sort of a chronic level of just low inflammation it was just constantly occurring now the best way to sort of give an analogy of this is it’s like they didn’t have any stress they never had a chance to actually grow and proliferate they were sheltered all the time you see em tor allows the growth so in hepatocyte your liver cells weren’t able to actually experience growth they remained these small sheltered cells that actually ended up having more stress in life because they weren’t able to handle things now the analogy that one of my team members actually gave me was like it sounded like someone that was possibly home schooled but never got the chance to ever have any social activity like okay so they were sheltered and they were never able to socialize like they’re too sheltered and then they grew up and their life was stressful because they never had any stress or experience or learned how to socialize right so that’s a perfect example of what happens if your employ levels are too low all the time so it’s exactly why we want to have them high and then low from time to time now let’s dive into the opposite of mTOR okay which is auto Fidji now auto Fidji is the cellular recycling now I’ve kind of explained it in videos before but sometimes I’m not really clear it’s not like cells literally eating each other it’s cells sort of recycling the components to become stronger so basically you have these cells and on top adji occurs when you’re in a time of stress like when you’re fasting and your cells say uh-oh we better start eating ourselves and recycling our own components so that we don’t have to start eating muscle tissue basically rather than a cell going out and eating muscle tissue and making you lose muscle first it’s first line of defense is to eat its own body parts that aren’t really being utilized so it recycles components eats them digests them recycles them you end up with a stronger more efficient cell so again to put this into context with mTOR when mTOR goes down autopsy goes up and vice versa now here’s what’s interesting is when you’re fasting amino acids trigger in tor so i’m digressing a little bit but i’ve talked about in videos how amino acids during a fast are dangerous and people always kind of give me crap about it right they always talk to me and say oh it’s like that’s such nonsense you’re getting way too way too granular here well amino acids trigger mTOR that’s defeating the purpose of the auto feed you don’t normally get one too fast so case in point here don’t have amino acids when you fast but again topic for a different day so one of the cool ways that I want you to look at this is like a bulk and a cut for yourselves now for those of you that are kind of familiar with the whole Fitness thought process of bulking and then cutting it’s like that for yourself so when you have mTOR activation with an excess of calories and working out the triggers mTOR you have your cells sort of bulking I mean they’re dividing they’re growing your time of excess so think of it like a bulk and think of autophagy like a cut it’s like you repeat that cycle in balance and you end up with a much stronger body it’s like you’re bulking you’re growing more muscle cells and then you have the action while toppity process that’s trimming them down and making them stronger you repeat rinse repeat rinse repeat and next thing you know you’ve got a bigger stronger leaner yet more efficient and longevity driven healthy cell it’s exactly what we want we want to be bigger stronger faster but also live for a long time so getting down to the nitty-gritty of it fasting increases auto Fujii tremendously but working out triggers mTOR tremendously so it actually works out if you work out in a fasted state you have a big spike of mTOR that is quickly recovered by a big spike in on top of you that occurs after the workout so although during your fasting period your workout will technically down regulate autophagy you’re doing yourself a huge service because you’re triggering mTOR without excess calories working out literally triggers imp tor okay so you’re doing that without excess calories you’re triggering this anabolic switch without having to have a bunch of food so it’s like right after that you start burning fat because on top edge is occurring and because you’re actually going into that fasted State so you’re literally getting the best of both worlds you’re triggering anabolism but then you’re triggering cell cleanup all while being in a calorie deficit that is pretty cool now the other thing is when you break your fast you’re going back into mTOR abundance you’re allowing your body to grow again and because insulin sensitivity is high import activation is going to be even higher so you have an extreme effect of streamium for activation followed by extreme town regulation and up regulation of auto fuji so that’s exactly what we want at the end of the day we’re getting balanced but we’re getting big spikes that trigger muscle growth and then trigger clean up muscle fat loss and longevity the trifecta everything we could possibly want okay so if it comes down to getting more information on fasting you know where to put your questions I want you to put your questions and any comments concerns down in the comment section below and I’ll try to answer those questions in my weekly round-up videos as always keep it locked in here in my channel and I’ll see you in the next video

This Post Was All About Does Exercise STOP the Effect of Fasting.
Does Exercise STOP the Effect of Fasting

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Does Exercise STOP the Effect of Fasting – Thomas DeLauer

mTOR

mTOR responds to signals from nutrients, growth factors, and cellular energy status and controls cell growth and proliferation based on this (by regulating protein synthesis)

In other words, whenever we have an increase in nutrition (mainly protein) and calories we essentially tell the body that plentiful times are here – our cells increase their working capacity and ATP production is increased

Cells increase division and we are then primed for growth and repair – mTOR is the protein that senses this

It’s important to realize that mTOR is neither good nor bad, but too much or too little can have negative consequences

You see, mTOR increases energy production, but also creates more junk products

Autophagy is the process that degrades these junk products, but it is only usually active when mTOR is decreased

The Good & Bad of mTOR Activation

The Good

mTOR activation allows us to put on more muscle (and fat) and we
increase various hormones such as IGF-1

mTOR increases ATP production and creates new mitochondria – it also increases mitochondrial metabolism (by activating PGC1a)

The Bad

Too much mTOR activation, however, contributes to a large number of human diseases, including cancer, obesity, type 2 diabetes, depression and neurodegeneration

Balance

A study published in the journal Cell Metabolism found that too little mTOR activation can be equally as bad as too much

The study found (in rats) that too little mTOR activity actually harms the liver – mice were fed an excess calorie diet, but given rapamycin to induce autophagy

Researchers found that persistent suppression of mTORC1 activity in the liver leads to low-level inflammation that is enhanced by feeding a HFD

Hepatocytes bearing deficient mTORC1 activity are less competent to cope with oxidative stress and other inflammation-related insults

Note: HCC is the fifth most common cancer subtype worldwide, with a median survival of 6–16 months after diagnosis

Persistent elevation of hepatic mTORC1 activity in the setting of overnutrition and obesity, or conversely, protracted suppression of mTORC1 activity induced by rapamycin and related drugs, sets in motion a spiraling sequence of pathologic events that promotes HCC.

Autophagy

The opposite side of the mTOR coin is the repair and recycling system – it’s activated with the mTOR switch is turned off

And, as we know, the main process that carries out repair and recycling in our cells is called autophagy

Autophagy is the mechanism our cells use to recycle damaged proteins and cell machinery (including mitochondria) and use their parts to make new machinery and new sources of energy – recycling old cellular machinery helps protect a cell from premature aging

Autophagy allows your cells to recycle used material for use as energy during stresses such as fasting, instead of breaking down valuable things such as muscle – preventing catabolism (breaking down) of muscle is always a good thing

Autophagy occurs at a low basal level in virtually all cells, being important in protein and organelle turnover – however, it may be up-regulated in times of need

When mTOR goes up, it shuts down autophagy – mTOR is sensitive to dietary amino acids (protein)

Fasting & Working Out

Journal – Autophagy

Fasting increase autophagy (as does caloric restriction and certain foods; EGCG, ginger, curcumin, etc.)

Explains that resistance training activates mTOR (as does protein, excess calories, IL-6, testosterone, etc.)

References

1) Jung CH , et al. (n.d.). mTOR regulation of autophagy. – PubMed – NCBI. Retrieved from
2) mTOR Pathways in Cancer and Autophagy. (n.d.). Retrieved from
3) Glick D , et al. (n.d.). Autophagy: cellular and molecular mechanisms. – PubMed – NCBI. Retrieved from
4) Autophagy fights disease through cellular self-digestion. (28, February). Retrieved from
5) Autophagy fights disease through cellular self-digestion. (28, February). Retrieved from
6) Resistance exercise initiates mechanistic target of rapamycin (mTOR) translocation and protein complex co-localisation in human skeletal muscle. (n.d.). Retrieved from
7) Short-term fasting induces profound neuronal autophagy. (16, August). Retrieved from /

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